Developmental Psychobiology Journal Impact Factor & Information

Publisher: International Society for Developmental Psychobiology, Wiley

Journal description

Developmental Psychobiology is a peer-reviewed journal that publishes original research papers from the disciplines of psychology biology neuroscience and medicine that contribute to an understanding of behavior development. Research that focuses on development in the embryo/fetus neonate juvenile or adult animal and multidisciplinary research that relates behavioral development to anatomy physiology biochemistry genetics or evolution is appropriate. The journal represents a broad phylogenetic perspective on behavior development by publishing studies of invertebrates fish birds humans and other animals. The journal publishes experimental and descriptive studies whether carried out in the laboratory or field. The journal also publishes review articles and theoretical papers that make important conceptual contributions. Special dedicated issues of Developmental Psychobiology consisting of invited papers on a topic of general interest may be arranged with the Editor-in-Chief. Developmental Psychobiology also publishes Letters to the Editor which discuss issues of general interest or material published in the journal. Letters discussing published material may correct errors provide clarification or offer a different point of view. Authors should consult the editors on the preparation of these contributions. Overall scholarship including soundness of experimental design appropriate controls and procedures and importance and significance are the major criteria for publication. Developmental Psychobiology is the official publication of the International Society for Developmental Psychobiology. Membership in the Society is not a prerequisite for submission or publication.

Current impact factor: 3.16

Impact Factor Rankings

2015 Impact Factor Available summer 2015
2013 / 2014 Impact Factor 3.163
2012 Impact Factor 2.595
2011 Impact Factor 2.977
2010 Impact Factor 2.062
2009 Impact Factor 2.119
2008 Impact Factor 1.891
2007 Impact Factor 1.872
2006 Impact Factor 1.946
2005 Impact Factor 1.583
2004 Impact Factor 2.083
2003 Impact Factor 1.712
2002 Impact Factor 1.371
2001 Impact Factor 1.286
2000 Impact Factor 1.322
1999 Impact Factor 1.312
1998 Impact Factor 1.596
1997 Impact Factor 1.435
1996 Impact Factor 1.203
1995 Impact Factor 1.041
1994 Impact Factor 1.421
1993 Impact Factor 1.188
1992 Impact Factor 1.144

Impact factor over time

Impact factor
Year

Additional details

5-year impact 2.48
Cited half-life 9.70
Immediacy index 0.66
Eigenfactor 0.01
Article influence 0.85
Website Developmental Psychobiology website
Other titles Developmental psychobiology (Online), Developmental psychobiology
ISSN 1098-2302
OCLC 38866749
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

Wiley

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • On author's personal website, institutional repositories, arXiv, AgEcon, PhilPapers, PubMed Central, RePEc or Social Science Research Network
    • Author's pre-print may not be updated with Publisher's Version/PDF
    • Author's pre-print must acknowledge acceptance for publication
    • Non-Commercial
    • Publisher's version/PDF cannot be used
    • Publisher source must be acknowledged with citation
    • Must link to publisher version with set statement (see policy)
    • If OnlineOpen is available, BBSRC, EPSRC, MRC, NERC and STFC authors, may self-archive after 12 months
    • If OnlineOpen is available, AHRC and ESRC authors, may self-archive after 24 months
    • Publisher last contacted on 07/08/2014
    • This policy is an exception to the default policies of 'Wiley'
  • Classification
    ​ yellow

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Stress influences a wide variety of outcomes including cognitive processing. In the rat, early life maternal care can influence developing offspring to affect both stress reactivity and cognitive processes in adulthood. The current study assessed if variations in early life maternal care can influence cognitive performance on a task, the ability to switch cognitive sets, dependent on the medial prefrontal cortex. Early in life, offspring was reared under High or Low maternal Licking conditions. As adults, they were trained daily and then tested on an attentional set-shifting task (ASST), which targets cognitive flexibility in rodents. Stress-sensitive behavioral and neural markers were assayed before and after the ASST. High and Low Licking offspring performed equally well on the ASST despite initial, but not later, differences in stress axis functioning. These results suggest that early life maternal care does not impact the accuracy of attentional set-shifting in rats. These findings may be of particular importance for those interested in the relationship between early life experience and adult cognitive function. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 08/2015; DOI:10.1002/dev.21343
  • [Show abstract] [Hide abstract]
    ABSTRACT: Polyvagal theory suggests that parasympathetic regulation of cardiac function, indexed by resting respiratory sinus arrhythmia (RSA), may be a marker of emotion regulatory capacity and associated with youth psychopathology. Contemporary models of psychopathology suggest that the effects of biological vulnerability may be moderated by developmental context. The aim of the present study was to examine whether parenting, particularly parental responses to youth's negative emotions, moderated the effects of resting RSA on depressive symptoms among early adolescents. We examined resting RSA, depressive symptoms, and parental responses to youth negative emotions among 120 adolescents aged 11-14 years (M = 12.86, SD = .85; 52.5% female). Resting RSA and lack of supportive parenting interacted to predict youth depressive symptoms, such that low resting RSA predicted more depressive symptoms only in the context of low levels of supportive parental responses to youth's negative emotions. By contrast, high resting RSA buffered the effects of low supportive parenting on youth depressive symptoms. These findings highlight the importance of understanding joint contributions of biological vulnerability and developmental context on youth depression outcomes. © 2015 Wiley Periodicals, Inc. Dev Psychobiol 9999: 1-10, 2015. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 08/2015; DOI:10.1002/dev.21347
  • [Show abstract] [Hide abstract]
    ABSTRACT: Numerous studies demonstrate that the Methionine variant of the catechol-O-methyltransferase Val158Met polymorphism, which confers less efficient catabolism of catecholamines, is associated with increased focal activation of prefrontal cortex (PFC) and higher levels of executive function abilities. By and large, however, studies of COMT Val158Met have been conducted with adult samples and do not account for the context in which development is occurring. Effects of early adversity on stress response physiology and the inverted U shape relating catecholamine levels to neural activity in PFC indicate the need to take into account early experience when considering relations between genes such as COMT and executive cognitive ability. Consistent with this neurobiology, we find in a prospective longitudinal sample of children and families (N = 1292) that COMT Val158Met interacts with early experience to predict executive function abilities in early childhood. Specifically, the Valine variant of the COMT Val158Met polymorphism, which confers more rather than less efficient catabolism of catecholamines is associated with higher executive function abilities at child ages 48 and 60 months and with faster growth of executive function for children experiencing early adversity, as indexed by cumulative risk factors in the home at child ages 7, 15, 24, and 36 months. Findings indicate the importance of the early environment for the relation between catecholamine genes and developmental outcomes and demonstrate that the genetic moderation of environmental risk is detectable in early childhood. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 08/2015; DOI:10.1002/dev.21332
  • [Show abstract] [Hide abstract]
    ABSTRACT: This paper revisits group difference and individual variability in birth weight, head size, Apgar score, and motor performance in neonatal and 8-month-old males and females using a large existing data set. The goal is primarily theoretical-to reframe existing analyses with an eye toward designing and executing more predictive analyses in the future. 3D graphing to visualize both the areas of overlap and regions of disparity between boys and girls has been used. A two-step cluster analysis of boys and girls together revealed three clusters. One was almost equally divided between boys and girls, but a second was highly enriched for boys and the third highly skewed toward girls. The relationship between cluster membership and Bayley motor scores at 8 months tested the hypothesis that initial differences that have no sex-related behavioral content might start processes that produce later sex-related differences. Initially, parental belief systems may be less important than infant care patterns evoked by basic size and health characteristics, even though later parental behaviors assume a decidedly gendered pattern. © 2015 Wiley Periodicals, Inc. Dev Psychobiol 9999: 1-12, 2015. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 08/2015; DOI:10.1002/dev.21345
  • [Show abstract] [Hide abstract]
    ABSTRACT: Socio-emotional processing is an essential part of development, and age-related changes in its neural correlates can be observed. The late positive potential (LPP) is a measure of motivated attention that can be used to assess emotional processing; however, changes in the LPP elicited by emotional faces have not been assessed across a wide age range in childhood and young adulthood. We used an emotional face matching task to examine behavior and event-related potentials (ERPs) in 33 youth aged 7-19 years old. Younger children were slower when performing the matching task. The LPP elicited by emotional faces but not control stimuli (geometric shapes) decreased with age; by contrast, an earlier ERP (the P1) decreased with age for both faces and shapes, suggesting increased efficiency of early visual processing. Results indicate age-related attenuation in emotional processing that may stem from greater efficiency and regulatory control when performing a socio-emotional task. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 07/2015; DOI:10.1002/dev.21341
  • Developmental Psychobiology 07/2015; DOI:10.1002/dev.21334
  • [Show abstract] [Hide abstract]
    ABSTRACT: Fetal alcohol spectrum disorder (FASD) is a devastating disease of the brain caused by exposure to alcohol during prenatal development. Its prevalence exceeds 1%. The majority of FASD cases represent the milder forms of the disease which often remain undiagnosed, and even when diagnosed treatment options for the patient are limited due to lack of information about the mechanisms that underlie the disease. The zebrafish has been proposed as a model organism for exploring the mechanisms of FASD. Our laboratory has been studying the effects of low doses of alcohol during embryonic development in the zebrafish. This review discusses the methods of alcohol exposure, its effects on behavioral performance including social behavior and learning, and the potential underlying biological mechanisms in zebrafish. It is based upon a recent keynote address delivered by the author, and it focuses on findings obtained mainly in his own laboratory. It paints a promising future of this small vertebrate in FASD research. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 06/2015; DOI:10.1002/dev.21318