Developmental Psychobiology Journal Impact Factor & Information

Publisher: International Society for Developmental Psychobiology, Wiley

Journal description

Developmental Psychobiology is a peer-reviewed journal that publishes original research papers from the disciplines of psychology biology neuroscience and medicine that contribute to an understanding of behavior development. Research that focuses on development in the embryo/fetus neonate juvenile or adult animal and multidisciplinary research that relates behavioral development to anatomy physiology biochemistry genetics or evolution is appropriate. The journal represents a broad phylogenetic perspective on behavior development by publishing studies of invertebrates fish birds humans and other animals. The journal publishes experimental and descriptive studies whether carried out in the laboratory or field. The journal also publishes review articles and theoretical papers that make important conceptual contributions. Special dedicated issues of Developmental Psychobiology consisting of invited papers on a topic of general interest may be arranged with the Editor-in-Chief. Developmental Psychobiology also publishes Letters to the Editor which discuss issues of general interest or material published in the journal. Letters discussing published material may correct errors provide clarification or offer a different point of view. Authors should consult the editors on the preparation of these contributions. Overall scholarship including soundness of experimental design appropriate controls and procedures and importance and significance are the major criteria for publication. Developmental Psychobiology is the official publication of the International Society for Developmental Psychobiology. Membership in the Society is not a prerequisite for submission or publication.

Current impact factor: 3.16

Impact Factor Rankings

2015 Impact Factor Available summer 2015
2013 / 2014 Impact Factor 3.163
2012 Impact Factor 2.595
2011 Impact Factor 2.977
2010 Impact Factor 2.062
2009 Impact Factor 2.119
2008 Impact Factor 1.891
2007 Impact Factor 1.872
2006 Impact Factor 1.946
2005 Impact Factor 1.583
2004 Impact Factor 2.083
2003 Impact Factor 1.712
2002 Impact Factor 1.371
2001 Impact Factor 1.286
2000 Impact Factor 1.322
1999 Impact Factor 1.312
1998 Impact Factor 1.596
1997 Impact Factor 1.435
1996 Impact Factor 1.203
1995 Impact Factor 1.041
1994 Impact Factor 1.421
1993 Impact Factor 1.188
1992 Impact Factor 1.144

Impact factor over time

Impact factor
Year

Additional details

5-year impact 2.48
Cited half-life 9.70
Immediacy index 0.66
Eigenfactor 0.01
Article influence 0.85
Website Developmental Psychobiology website
Other titles Developmental psychobiology (Online), Developmental psychobiology
ISSN 1098-2302
OCLC 38866749
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

Wiley

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • On author's personal website, institutional repositories, arXiv, AgEcon, PhilPapers, PubMed Central, RePEc or Social Science Research Network
    • Author's pre-print may not be updated with Publisher's Version/PDF
    • Author's pre-print must acknowledge acceptance for publication
    • On a non-profit server
    • Publisher's version/PDF cannot be used
    • Publisher source must be acknowledged with citation
    • Must link to publisher version with set statement (see policy)
    • If OnlineOpen is available, BBSRC, EPSRC, MRC, NERC and STFC authors, may self-archive after 12 months
    • If OnlineOpen is available, AHRC and ESRC authors, may self-archive after 24 months
    • Publisher last contacted on 07/08/2014
    • This policy is an exception to the default policies of 'Wiley'
  • Classification
    ​ yellow

Publications in this journal

  • Developmental Psychobiology 05/2015; 57(4). DOI:10.1002/dev.21314
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    ABSTRACT: Prenatal exposure to beverage alcohol is a major cause of mild mental retardation and developmental delay. In nonendangered alcohol-preferring vervet monkeys, we modeled the most common nondysmorphic form of fetal alcohol syndrome disorder with voluntary drinking during the third trimester of pregnancy. Here, we report significant numerical reductions in the principal hippocampal neurons of fetal alcohol-exposed (FAE) offspring, as compared to age-matched, similarly housed conspecifics with isocaloric sucrose exposure. These deficits, particularly marked in CA1 and CA3, are present neonatally and persist through infancy (5 months) and juvenile (2 years) stages. Although the volumes of hippocampal subdivisions in FAE animals are not atypical at birth, by age 2, they are only 65-70% of those estimated in age-matched controls. These data suggest that moderate, naturalistic alcohol consumption during late pregnancy results in a stable loss of hippocampal neurons and a progressive reduction of hippocampal volume. © 2015 The Authors. Developmental Psychobiology Published by Wiley Periodicals, Inc. Dev Psychobiol 57:470𠄃485, 2015. © 2015 The Authors. Developmental Psychobiology Published by Wiley Periodicals, Inc.
    Developmental Psychobiology 05/2015; 57(4). DOI:10.1002/dev.21311
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    ABSTRACT: Young adult survivors of extremely low birth weight (ELBW; <1000 g) are known to be at elevated risk for internalizing problems, though little is known about the mechanisms that may lead to higher levels of psychopathology in this vulnerable group. We examined the moderating influence of neuroendocrine functioning on the link between being born at ELBW and internalizing behaviors at age 30-35. Salivary cortisol was collected 20 min after completion of a social stress task in 83 ELBW adult survivors and 89 normal birth weight (NBW; >2500 g) controls. Using a median split, participants were separated into two groups (high or low afternoon cortisol levels). ELBW survivors with "high" afternoon cortisol levels self-reported significantly higher levels of internalizing behaviors compared to those with "low" afternoon cortisol levels. This association between afternoon cortisol and internalizing symptoms did not exist among NBW controls. These results are suggestive of a differential susceptibility for internalizing behaviors among ELBW survivors, depending on their ability to regulate neuroendocrine responses. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21308
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    ABSTRACT: Reactivation is an automatic, perceptual process in which exposure to components of a forgotten event alleviates forgetting. Most research on infant memory reactivation has used conditioning paradigms. We used the puppet imitation task to systematically examine which stimuli could retrieve 6-month-olds' forgotten memory of the modeled actions. Infants watched an adult model a sequence of actions on a puppet, imitated the actions, and were exposed to reactivation cues 24 hr before a 7-day (Experiment 1) or 14-day (Experiment 2) retention test. Exposure to any component of the original event reactivated the memory during the 7-day test, but two of the same components failed to alleviate forgetting during the 14-day test. Increasing the number of retrieval cues facilitated 14-day test performance. These findings reveal that the principles of reactivation are the same for conditioning and imitation paradigms: The necessary and sufficient conditions for memory reactivation are directly related to memory accessibility. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21298
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    ABSTRACT: Despite extensive examination of episodic memory and future thinking development, little is known about the concurrent emergence of these capacities during early childhood. In Experiment 1, 3-year-olds participated in an episodic memory hiding task ("what, when, where" [WWW] components) with an episodic future thinking component. In Experiment 2, a group of 4-year-olds (including children from Experiment 1) participated in the same task (different objects and locations), providing the first longitudinal investigation of episodic memory and future thinking. Although children exhibited age-related improvements in recall, recognition, and binding of the WWW episodic memory components, there were no age-related changes in episodic future thinking. At both ages, WWW episodic memory performance was higher than future thinking performance, and episodic future thinking and WWW memory components were unrelated. These findings suggest that the WWW components of episodic memory are potentially less fragile than the future components when assessed in a cognitively demanding task. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21307
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    ABSTRACT: Recent evidence has revealed unique patterns of behavioral development after prenatal insult similar to those outlined in studies of adult metabolic dysfunction after prenatal malnutrition. The hallmark features of this Developmental Pathway include a prenatal insult to the nervous system (environmental or genetic) followed by a period of Silent Vulnerability, where no or few functional deficits are observed, and finally emergence of later dysfunction. Possible mechanisms leading to later dysfunction from prenatal insult may include secondary or cascade effects due to the timing of prenatal insults relative to later developing structures in the brain. Methods best employed to study the mechanisms of these pathways are microgenetic and longitudinal designs that include behavioral assessment during the prenatal period of development, and animal models such as the guinea pig. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21304
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    ABSTRACT: In an earlier study of newly hatched chicks we reported that continuous bright light exposure throughout incubation accelerated locomotor development and continuous dark exposure delayed it, compared to less intense, intermittent light exposure. Commonly studied gait parameters indicated locomotor skill was similar across groups. However, dark incubated chicks walked with a greater step width, raising the possibility of differences in dynamic balance and control of forward progression. In this study, we established methods to retrospectively examine the previously published locomotor data for differences in lateral drift. We hypothesized that chicks incubated in darkness would exhibit more drift than chicks incubated in light. Analyses identified differences in forward progression between chicks incubated in the two extreme light conditions, supporting the study's hypothesis. We discuss the significance of our findings and potential design considerations for future studies of light-accelerated motor development in precocial and nonprecocial animals. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21306
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    ABSTRACT: We tested whether respiratory sinus arrhythmia (RSA) reactivity in response to each of three self-regulation tasks (bird and dragon; knock-tap; and gift wrap) would predict self-regulation performance in a sample of 101 preschool-age children (M age = 4.49, SD = .64). While controlling for baseline RSA, decreases in RSA from bird and dragon to knock-tap (but not from baseline to bird and dragon) predicted a latent variable measuring self-regulation. Furthermore, increases in RSA from the knock-tap to gift wrap—the only task involving delay of gratification—were related to concurrent task performance while controlling for the relation between RSA reactivity and the latent self-regulation variable. Results suggest that the relations between RSA reactivity and self-regulatory ability are influenced by task-specific demands and possibly by task order. Furthermore, RSA reactivity appears to relate differently to performance on motivationally salient self-regulation tasks such as delay of gratification relative to cool executive function tasks. © Dev Psychobiol
    Developmental Psychobiology 04/2015; DOI:10.1002/dev.21315
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    ABSTRACT: The human prefrontal cortex, important for executive functions, loses gray matter throughout the adolescent period. In rats, our laboratory demonstrated that a loss of neurons between adolescence and adulthood partially underlies the loss of volume, and this loss is greater in females than males. Here, we examine whether being deprived of gonadal hormones before puberty through adulthood influences the number of neurons in the medial prefrontal cortex (mPFC). Prior to puberty, the testes or ovaries were removed in male and female rats. In adulthood, the number of neurons and glia in the mPFC were quantified using unbiased stereology, and the volume of the frontal white matter was measured. Prepubertal ovariectomy resulted in a higher number of neurons and glia and a larger volume of white matter compared to sham control littermates. Castrated males were not different from sham males on any measure. Thus ovarian hormones secreted after puberty influence the cellular composition of the medial prefrontal cortex. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 03/2015; 57(3). DOI:10.1002/dev.21290
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    ABSTRACT: An infant-controlled tactile habituation without visual control procedure was used to evaluate the ability of 32 late-preterm neonates (mean gestational age: 34 weeks) and 32 early-term neonates (mean gestational age: 38 weeks) to actively explore with hands objects varying in texture (smooth, granular). Holding time and Hand Pressure Frequency (HPF) were recorded. Holding time decreased as habituation progressed in both group of neonates. Holding time increased from habituation trials to test trials only in early-term neonates. A reaction to novelty was only observed in early-term neonates. During habituation, HPF remained unchanged in late-preterm infants whereas HPF decreased in early-term infants. HPF increased from habituation trials to test trials in early-term neonates and in late-preterm infants. However, reaction to novelty was only observed for early-term infants. The significance of these results is discussed in reference to brain maturation in preterm infants. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 03/2015; 57(3). DOI:10.1002/dev.21295
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    ABSTRACT: The biological basis of parenting has received recent attention given the profound effects of parenting on both child and parent health outcomes. This study examined the moderating role of child temperamental effortful control on the association between observed parental hostility and parents' cortisol awakening response (CAR), a critical index of stress system functioning. Participants included 149 parents and their preschool-aged children. Parents obtained salivary cortisol samples at waking, and 30 and 45 min post-waking across two consecutive days. Parental hostility was assessed during an observational parent-child interaction task, and child effortful control was assessed using parent report. Parental hostility was associated with parents' lower cortisol levels at 30 and 45 min post-waking and lower CAR. Moreover, results demonstrated an interaction between parenting and child temperament on parent CAR. The findings highlight the need to examine the interplay between parenting and child temperament on parents' stress physiology. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 03/2015; 57(3). DOI:10.1002/dev.21301
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    ABSTRACT: Prenatal stress (PNS) is a risk factor for the development of neuropsychiatric disorders. This study was aimed at assessing, in a rodent model, changes in gene expression profiles and behavioral output as a result of PNS, during periadolescence, a critical developmental period for the onset of psychopathology. Social behavior was studied in a standardized social interaction paradigm and the expression of Brain-Derived Neurotrophic Factor (Bdnf), a marker of neuronal plasticity, and of inhibitory and excitatory mechanisms (Na(+) -K(+) -2Cl(-) and K(+) -Cl(-) cotransporters ratio, NKCC1/KCC2) was analyzed. Results indicate that PNS reduced Bdnf transcripts while increasing the NKCC1/KCC2 ratio, primarily in the hippocampus. In the prefrontal cortex, changes in Bdnf were found to be gender-dependent. These effects were accompanied by reduced levels of affiliative and investigative social behaviors. Interestingly, interaction with non-stressed subjects was able to improve sociality in PNS rats suggesting that the social environment could be exploited for therapeutic intervention. © 2015 Wiley Periodicals, Inc. Dev Psychobiol. © 2015 Wiley Periodicals, Inc.
    Developmental Psychobiology 03/2015; 57(3). DOI:10.1002/dev.21297