Hearing Research (HEARING RES)

Publisher: Elsevier

Journal description

The aim of the journal is to provide a forum for papers concerned with basic auditory mechanisms. Emphasis is on experimental studies, but theoretical papers will also be considered. The editor of the journal is prepared to accept original research papers in the form of full-length papers, short communications, letters to the Editor, and reviews. Papers submitted should deal with auditory neurophysiology, ultrastructure, psychoacoustics and behavioural studies of hearing in animals, and models of auditory functions. Papers on comparative aspects of hearing in animals and man, and on effects of drugs and environmental contaminants on hearing function will also be considered. Clinical papers will not be accepted unless they contribute to the understanding of normal hearing functions.

Current impact factor: 2.97

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 2.968
2013 Impact Factor 2.848
2012 Impact Factor 2.537
2011 Impact Factor 2.696
2010 Impact Factor 2.428
2009 Impact Factor 2.177
2008 Impact Factor 2.333
2007 Impact Factor 2.062
2006 Impact Factor 1.584
2005 Impact Factor 1.674
2004 Impact Factor 1.578
2003 Impact Factor 1.502
2002 Impact Factor 1.969
2001 Impact Factor 1.586
2000 Impact Factor 1.753
1999 Impact Factor 1.804
1998 Impact Factor 1.598
1997 Impact Factor 1.915
1996 Impact Factor 1.641
1995 Impact Factor 1.908
1994 Impact Factor 1.744
1993 Impact Factor 1.853
1992 Impact Factor 1.792

Impact factor over time

Impact factor

Additional details

5-year impact 3.14
Cited half-life >10.0
Immediacy index 1.24
Eigenfactor 0.01
Article influence 1.07
Website Hearing Research website
Other titles Hearing research
ISSN 0378-5955
OCLC 4410062
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Authors pre-print on any website, including arXiv and RePEC
    • Author's post-print on author's personal website immediately
    • Author's post-print on open access repository after an embargo period of between 12 months and 48 months
    • Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months
    • Author's post-print may be used to update arXiv and RepEC
    • Publisher's version/PDF cannot be used
    • Must link to publisher version with DOI
    • Author's post-print must be released with a Creative Commons Attribution Non-Commercial No Derivatives License
    • Publisher last reviewed on 03/06/2015
  • Classification

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Binaural interaction in the auditory brainstem response (ABR) represents the discrepancy between the binaural waveform and the sum of monaural ones. A typical ABR binaural interaction in humans is a reduction of the binaural amplitude compared to the monaural sum at the wave-V latency, i.e., the DN1 component. It has been considered that the DN1 is mainly elicited by high frequency components of stimuli whereas some studies have shown the contribution of low-to-middle frequency components to the DN1. To examine this issue, the present study compared the ABR binaural interaction elicited by tone pips (1 kHz, 10-ms duration) with the one by clicks (a rectangular wave, 0.1-ms duration) presented at 80 dB peak equivalent SPL and a fixed stimulus onset interval (180 ms). The DN1 due to tone pips was vulnerable compared to the click-evoked DN1. The pip-evoked DN1 was significantly detected under auditory attention whereas it failed to reach significance under visual attention. The click-evoked DN1 was robustly present for the two attention conditions. The current results might confirm the high frequency sound contribution to the DN1 elicitation. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 03/2015; 325. DOI:10.1016/j.heares.2015.02.010
  • [Show abstract] [Hide abstract]
    ABSTRACT: As frequency is one of the most basic elements of sound, it is not surprising that the earliest stages of auditory cortical processing are tonotopically organized. In cats, there are four known tonotopically organized cortical areas: the anterior (AAF), posterior (PAF), and ventral posterior (VPAF) auditory fields and primary auditory cortex (A1). Electrophysiological and anatomical evidence have suggested that AAF and A1 form core auditory cortex. The purpose of this investigation was to determine if high-field functional magnetic resonance imaging (fMRI) could be used to define the borders of all four tonotopically organized areas, identify core auditory cortex, and demonstrate tonotopy similar to that found using more invasive techniques. Five adult cats were examined. Eight different pure tones or one broad-band noise (BBN) stimuli were presented in a block paradigm during continuous fMRI scanning. Analysis was performed on each animal individually using conservative familywise error thresholds. Group analysis was performed by extracting data from fMRI analysis software and performing a battery of statistical tests. In auditory cortex, a reversal of the tonotopic gradient is known to occur at the borders between tonotopically organized areas. Therefore, high and low tones were used to delineate these borders. Activations in response to BBN as opposed to tonal stimulation demonstrated that core auditory cortex consists of both A1 and AAF. Finally, tonotopy was identified in each of the four known tonotopically organized areas. Therefore, we conclude that fMRI is effective at defining all four tonotopically organized cortical areas and delineating core auditory cortex. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 03/2015; 325. DOI:10.1016/j.heares.2015.03.003
  • [Show abstract] [Hide abstract]
    ABSTRACT: The central auditory system produces combination-sensitive neurons tuned to a specific combination of multiple signal elements. Some of these neurons act as coincidence detectors with delay lines for the extraction of spectro-temporal information from sounds. "Delay-tuned" neurons of mustached bats are tuned to a combination of up to four signal elements with a specific delay between them and form a delay map. They are produced in the inferior colliculus by the coincidence of the rebound response following glycinergic inhibition to the first harmonic of a biosonar pulse with the short-latency response to the 2nd-3rd harmonics of its echo. Compared with collicular delay-tuned neurons, thalamic and cortical ones respond more to pulse-echo pairs than individual sounds. Cortical delay-tuned neurons are clustered in the three separate areas. They interact with each other through a circuit mediating positive feedback and lateral inhibition for adjustment and improvement of the delay tuning of cortical and subcortical neurons. The current article reviews the mechanisms for delay tuning and the response properties of collicular, thalamic and cortical delay-tuned neurons in relation to hierarchical signal processing. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 03/2015; 5. DOI:10.1016/j.heares.2015.02.008
  • Source

    Hearing Research 02/2015; 320. DOI:10.1016/j.heares.2014.11.004
  • [Show abstract] [Hide abstract]
    ABSTRACT: Older adults, even with clinically normal hearing sensitivity, often report difficulty understanding speech in the presence of background noise. Part of this difficulty may be related to age-related degradations in the neural representation of speech sounds, such as formant transitions. Frequency-following responses (FFRs), which are dependent on phase-locked neural activity, were elicited using sounds consisting of linear frequency sweeps, which may be viewed as simple models of formant transitions. Eighteen adults (ten younger, 22 - 24 years old, and nine older, 51 - 67 years old) were tested. FFRs were elicited by tonal sweeps in six conditions. Two directions of frequency change, rising or falling, were used for each of three rates of frequency change. Stimulus-to-response cross correlations revealed that older adults had significantly poorer representation of the tonal sweeps, and that FFRs became poorer for faster rates of change. An additional FFR signal-to-noise ratio analysis based on time windows revealed that across the FFR waveforms and rates of frequency change, older adults had smaller (poorer) signal-to-noise ratios. These results indicate that older adults, even with clinically-normal hearing sensitivity, have degraded phase-locked neural representations of dynamic frequency. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 02/2015; 323. DOI:10.1016/j.heares.2015.02.002
  • [Show abstract] [Hide abstract]
    ABSTRACT: Human sound source localization relies on various acoustical cues one of the most important being the interaural time difference (ITD). ITD is best detected in the fine structure of low-frequency sounds but it may also contribute to spatial hearing at higher frequencies if extracted from the sound envelope. The human brain mechanisms related to this envelope ITD cue remain unexplored. Here, we tested the sensitivity of the human auditory cortex to envelope ITD in magnetoencephalography (MEG) recordings. We found two types of sensitivity to envelope ITD. First, the amplitude of the auditory cortical N1m response was smaller for zero envelope ITD than for long envelope ITDs corresponding to the sound being in opposite phase in the two ears. Second, the N1m response amplitude showed ITD-specific adaptation for both fine-structure and for envelope ITD. The auditory cortical sensitivity was weaker for envelope ITD in high-frequency sounds than for fine-structure ITD in low-frequency sounds but occurred within a range of envelope ITDs that are encountered in natural conditions. Finally, the participants were briefly tested for their behavioral ability to detect envelope ITD. Interestingly, we found a correlation between the behavioral performance and the neural sensitivity to envelope ITD. In conclusion, our findings show that the human auditory cortex is sensitive to ITD in the envelope of high-frequency sounds and this sensitivity may have behavioral relevance. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 02/2015; 323. DOI:10.1016/j.heares.2015.01.014
  • [Show abstract] [Hide abstract]
    ABSTRACT: We recently reported that forward acoustic masking can enhance the auditory brainstem response (ABR) in rats treated with a high dose of sodium salicylate (NaSal), a tinnitus inducer, when tested in open acoustic field (Liu & Chen, 2012, Brain Research 1485, 88-94). In the present study, we first replicated this experiment in closed acoustic field under two conditions: (1) the forward masker and the probe were presented to both ears (diotic paradigm); (2) the forward masker was presented to one ear and the probe to the other ear (dichotic paradigm). We found that only when the stimuli were presented by using the diotic, rather than the dichotic, paradigm could forward acoustic masking enhance the ABR in the rat treated with NaSal (300 mg/kg). The enhancement was obvious for ABR waves II and IV, but not for wave I, indicating a central origin. The enhancement occurred at the high frequencies (16, 24, 32 kHz) at which the animals demonstrated a tinnitus-like behavior as revealed by using the gap prepulse inhibition of acoustic startle paradigm. We then administered vigabatrin, a GABA transaminase inhibitor, in the animals to suppress NaSal-induced tinnitus. The vigabatrin treatment successfully prevented forward acoustic masking from enhancing the ABR. These findings demonstrate that the observed enhancement of ABRs by forward acoustic masking originates in the central auditory pathway ipsilateral to the stimulated ear. We propose that the enhancement is closely associated with NaSal-induced tinnitus. Copyright © 2015. Published by Elsevier B.V.
    Hearing Research 02/2015; 323. DOI:10.1016/j.heares.2015.01.013