International Journal of Obesity (INT J OBESITY)

Publisher: International Association for the Study of Obesity, Nature Publishing Group

Journal description

Multi-disciplinary forum for research describing: basic clinical and applied studies in biochemistry, physiology, genetics and nutrition, molecular, metabolic, psychological and epidemiological aspects of obesity and related disorders.

Current impact factor: 5.39

Impact Factor Rankings

2015 Impact Factor Available summer 2015
2013 / 2014 Impact Factor 5.386
2012 Impact Factor 5.221
2011 Impact Factor 4.691
2010 Impact Factor 5.125
2009 Impact Factor 4.343
2008 Impact Factor 3.64
2007 Impact Factor 3.56
2006 Impact Factor 4.055
2005 Impact Factor 4.482
2004 Impact Factor 3.459
2003 Impact Factor 2.794
2002 Impact Factor 2.363
2001 Impact Factor 2.196
2000 Impact Factor 2.982
1999 Impact Factor 3.199
1998 Impact Factor 3.003
1997 Impact Factor 2.476
1996 Impact Factor 1.759
1995 Impact Factor 1.832
1994 Impact Factor 1.568
1993 Impact Factor 1.776
1992 Impact Factor 1.607

Impact factor over time

Impact factor

Additional details

5-year impact 5.52
Cited half-life 8.00
Immediacy index 1.17
Eigenfactor 0.04
Article influence 1.85
Website International Journal of Obesity website
Other titles International journal of obesity
ISSN 0307-0565
OCLC 26074170
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details

Nature Publishing Group

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 6 months embargo
  • Conditions
    • Authors retain copyright
    • Published source must be acknowledged and DOI cited
    • Must link to publisher version
    • Publisher's version/PDF cannot be used
    • On author's personal website and institutional repository
    • If funding agency rules apply, authors may post authors version to their relevant funding body's archive, 6 months after publication
    • This policy is an exception to the default policies of 'Nature Publishing Group'
  • Classification
    ​ yellow

Publications in this journal

  • International Journal of Obesity 04/2015;
  • International Journal of Obesity 01/2015;
  • [Show abstract] [Hide abstract]
    ABSTRACT: Objective:This study aims to assess the natural course of metabolically healthy abdominal obesity (MHAO) phenotype and determine the predictors of change in the metabolic status in this population over 10 years of follow-up.Methods:A total of 916 MHAO subjects from Tehran Lipid and Glucose Study (TLGS) were followed for changes in their metabolic health status. Anthropometric and metabolic indices were measured at baseline and were compared between subjects with healthy and unhealthy metabolic conditions at the end of follow-up. Predictors of change in metabolic health were assessed in logistic regression models. National waist circumference cut-offs were used for definition of abdominal obesity. Metabolic health was defined as ⩽1 metabolic components of metabolic syndrome according to Joint Interim Statement criteria.Results:At the end of the follow-up nearly half of the MHAO subjects lost their metabolic health and 42.1% developed metabolic syndrome by definition. Low High Density Lipoprotein-Cholesterol (HDL-C), hypertriglyceridemia, and homeostasis model assessment-insulin resistance (HOMA-IR) at baseline were significant predictors of change in metabolic health condition.Conclusion:MHAO is a relatively unstable condition and a considerable percentage of these individuals will lose their metabolic health as time passes. Baseline metabolic characteristics may be useful predictors of this change and should be considered in the care of these individuals.International Journal of Obesity accepted article preview online, 07 October 2014. doi:10.1038/ijo.2014.176.
    International Journal of Obesity 10/2014; DOI:10.1038/ijo.2014.176
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    ABSTRACT: Background Elevated levels of intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) may contribute to cardiovascular disease and are associated with obstructive sleep apnea (OSA) and obesity. The relationship between OSA and obesity in determining ICAM-1 and VCAM-1 levels, and the effect of treatment, is unclear.Objective Our aim was to study whether positive airway pressure (PAP) usage resulted in changes in ICAM-1 and VCAM-1 after 2 years within 309 OSA patients from the Icelandic Sleep Apnea Cohort, and determine how obesity affected such changes.Subjects/Methods The mean body mass index (BMI) was 32.4±5.1 kg/m(2); subjects had moderate-to-severe OSA (apnea-hypopnea index=45.0±20.2) and 79% were male. There were 177 full PAP users (⩾4 hours/night and ⩾20 of last 28 nights), 44 partial (<4 hours/night or <20 nights), and 88 non-users.ResultsICAM-1 (P<0.001) and VCAM-1 (P=0.012) change was significantly different among the PAP groups. The largest ICAM-1 differences were among the most obese subjects (P<0.001). At follow-up, non-users had increased ICAM-1 compared to decreased levels in full users. All groups had increased VCAM-1, but non-users had a significantly larger increase than full users.Conclusion Within moderate-to-severe OSA patients, PAP usage prevents increases in adhesion molecules observed in non-users after two years. For ICAM-1, the largest effect is in the most obese subjects. As OSA and obesity commonly coexist, the usage of PAP to limit increases in adhesion molecules may decrease the rate of progression of OSA-related cardiovascular disease.International Journal of Obesity accepted article preview online, 21 July 2014; doi:10.1038/ijo.2014.123.
    International Journal of Obesity 07/2014; DOI:10.1038/ijo.2014.123
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    ABSTRACT: The central nervous melanocortin system maintains body mass and adiposity within a ‘healthy’ range by regulating satiety and metabolic homeostasis. Neural melanocortin-4 receptors (MC4R) modulate satiety signals and regulate autonomic outputs governing glucose and lipid metabolism in the periphery. The functions of melanocortin-3 receptors (MC3R) have been less well defined. We have observed that food anticipatory activity (FAA) is attenuated in Mc3r�/� mice housed in light:dark or constant dark conditions. Mc3r�/� mice subjected to the restricted feeding protocol that was used to induce FAA also developed insulin resistance, dyslipidaemia, impaired glucose tolerance and evidence of a cellular stress response in the liver. MC3Rs may thus function as modulators of oscillator systems that govern circadian rhythms, integrating signals from nutrient sensors to facilitate synchronizing peak foraging behaviour and metabolic efficiency with nutrient availability. To dissect the functions of MC3Rs expressed in hypothalamic and extra-hypothalamic structures, we inserted a ‘lox-stop-lox’ (TB) sequence into the Mc3r gene. Mc3rTB/TB mice recapitulate the phenotype reported for Mc3r�/� mice: increased adiposity, accelerated diet-induced obesity and attenuated FAA. The ventromedial hypothalamus exhibits high levels of Mc3r expression; however, restoring the expression of the LoxTB Mc3r allele in this nucleus did not restore FAA. However, a surprising outcome came from studies using Nestin-Cre to restore the expression of the LoxTB Mc3r allele in the nervous system. These data suggest that ‘non-neural’ MC3Rs have a role in the defence of body weight. Future studies examining the homeostatic functions of MC3Rs should therefore consider actions outside the central nervous system.
    International Journal of Obesity 07/2014; Supplements(4):S37-S44. DOI:10.1038/ijosup.2014.10
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    ABSTRACT: Depression has been associated with increased risk of cardiovascular disease. The inflammatory marker C-reactive protein (CRP) has also been identified as an independent predictor of short- and long-term cardiovascular disease events. Inflammation may influence the relationship between depression and cardiovascular disease.
    International Journal of Obesity 08/2013; 37:S38-S43. DOI:10.1038/ijo.2013.95
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    ABSTRACT: OBJECTIVE: This study investigated changes in the quality of life of men and women who participated in a primary care-based weight loss intervention program. METHODS: Participants were enrolled in a 2-year randomized clinical trial (POWER-UP) conducted at the University of Pennsylvania and in six affiliated primary care practices. Inclusion criteria included the presence of obesity (body mass index of 30-50 kg m(-2)) and at least two components of the metabolic syndrome. MAIN OUTCOME MEASURES: Quality of life was assessed by three measures: the Short Form Health Survey (SF-12); the Impact of Weight on Quality of Life-Lite; and the EuroQol-5D. RESULTS: Six months after the onset of treatment, and with a mean weight loss of 3.9 +/- 0.3 kg, participants reported significant improvements on all measures of interest with the exception of the Mental Component Score of the SF-12. These changes remained significantly improved from baseline to month 24, with the exception of the EuroQol-5D. Many of these improvements were correlated with the magnitude of weight loss and, for the most part, were consistent across gender and ethnic group. CONCLUSIONS: Individuals with obesity and components of the metabolic syndrome reported significant improvements in most domains of the quality of life with a modest weight loss of 3.7% of initial weight, which was achieved within the first 6 months of treatment. The majority of these improvements were maintained at month 24, when participants had lost 3.0% of their weight.
    International Journal of Obesity 08/2013; 37:S25-S30. DOI:10.1038/ijo.2013.93
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    ABSTRACT: To examine changes in eating behaviors and physical activity, as well as predictors of weight loss success, in obese adults who participated in a 2-year behavioral weight loss intervention conducted in a primary care setting.
    International Journal of Obesity 08/2013; 37:S12-S18. DOI:10.1038/ijo.2013.91