International Journal for Vitamin and Nutrition Research Impact Factor & Information

Publisher: Hogrefe

Journal description

Since 1930 this journal has provided an important international forum for scientific advances in the study of nutrition and vitamins. Widely read by academicians as well as scientists working in major governmental and corporate laboratories throughout the world, this publication presents work dealing with basic as well as applied topics. The editorial and advisory boards include many of the leading persons currently working in this area. The journal is of particular interest to: Nutritionists, Vitaminologists, Biochemists, Physicians, Engineers of human and animal nutrition.

Current impact factor: 0.85

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 0.854
2013 Impact Factor 1
2012 Impact Factor 1.271
2011 Impact Factor 0.877
2010 Impact Factor 0.74
2009 Impact Factor 0.897
2008 Impact Factor 0.971
2007 Impact Factor 0.738
2006 Impact Factor 0.862
2005 Impact Factor 1.034
2004 Impact Factor 1.071
2003 Impact Factor 1.019
2002 Impact Factor 0.883
2001 Impact Factor 1.175
2000 Impact Factor 1.299
1999 Impact Factor 1.083
1998 Impact Factor 0.949
1997 Impact Factor 0.594
1996 Impact Factor 0.764
1995 Impact Factor 0.698
1994 Impact Factor 0.418
1993 Impact Factor 0.627
1992 Impact Factor 0.453

Impact factor over time

Impact factor

Additional details

5-year impact 1.30
Cited half-life >10.0
Immediacy index 0.17
Eigenfactor 0.00
Article influence 0.34
Website International Journal for Vitamin and Nutrition Research website
Other titles International journal for vitamin and nutrition research, Internationale Zeitschrift für Vitamin- und Ernährungsforschung, Journal international de vitaminologie et de nutrition
ISSN 0300-9831
OCLC 1785670
Material type Periodical
Document type Journal / Magazine / Newspaper

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • On author's personal website, institutional website or institutional repository
    • On a non-profit server
    • Publisher's version/PDF cannot be used
    • Publisher copyright must be acknowledged with set statement (see policy)
    • Must link to DOI
    • Set phrase must appear "This article does not exactly replicate the final version published in the journal "[Add title of Journal]". It is not a copy of the original published article and is not suitable for citation."
    • Upon written request authors may archive on a website or in a repository mandated by their funding body 12 months after publication or in accordance with legal obligations funding bodies
  • Classification

Publications in this journal

  • International Journal for Vitamin and Nutrition Research 11/2015; DOI:10.1024/0300-9831/a000228

  • International Journal for Vitamin and Nutrition Research 09/2015;
  • Sakineh Shab Bidar · Tirang R. Neyestani · Abolghassem Djazayery ·

    International Journal for Vitamin and Nutrition Research 08/2015;

  • International Journal for Vitamin and Nutrition Research 07/2015; 85(1-2):31-38.

  • International Journal for Vitamin and Nutrition Research 07/2015; 85(1-2):31-38.

  • International Journal for Vitamin and Nutrition Research 06/2015; 85(1-2):50-60.
  • [Show abstract] [Hide abstract]
    ABSTRACT: Aim: The purpose of this study was to investigate the effects of curcumin on eccentric exerciseinduced muscle damage and oxidative stress in rats. Methods: Thirty male Wistar rats were divided into four groups: Control (C; no curcumin, no exercise; n = 6), Curcumin (Cur; n = 8), Exercise (E; n = 8) and Exercise Plus Curcumin (ECur; n = 8). Curcumin was given for 20 days via oral gavage at doses of 200 mg/kg-1 of body weight per day, dissolved in corn oil. On the 21st day eccentric exercise was provided via a treadmill run and the rats were sacrificed immediately after. Results: Eccentric exercise resulted in significant (p < 0.05) increases in all injury markers such as creatine kinase (CK) and myoglobin, but curcumin supplementation tended to decrease CK activity (p > 0.05) and significantly decreased myoglobin levels (p < 0.05). In blood and muscle samples, malondialdehyde (MDA) levels were not affected by either curcumin or exercise (p > 0.05). MDA levels in liver tissue decreased in the ECur group, compared to the control (p < 0.05). Superoxide dismutase (SOD) activities and glutathione (GSH) levels were affected by neither curcumin nor exercise (p > 0.05), in blood, muscle and liver tissues. Conclusion: The results of this study suggest that curcumin has a protective effect on eccentric exercise induced muscle damage, and that this effect might be independent of oxidative stress and antioxidant systems.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0163-0172. DOI:10.1024/0300-9831/a000203
  • [Show abstract] [Hide abstract]
    ABSTRACT: Animal and human studies indicate that omega (n)-3 polyunsaturated fatty acids (PUFA) can influence bone health. We conducted a randomized, double-blind, placebo-controlled trial of the effects of n-3 long chain (LC) PUFA supplementation (N-3 LCPUFA) on red blood cell (RBC) fatty acid levels and bone turnover markers in older postmenopausal women. One hundred and twenty-six postmenopausal women (mean age 75 ± 7 years) were treated with n-3 LCPUFA (1.2 g eicosapentaenoic acid [EPA]/docosahexaenoic acid [DHA]/day, n = 85) or placebo (olive oil, n = 41) for 6 months. All women received 315 mg calcium citrate and 1000 IU cholecalciferol. RBC DHA (weight %) increased in the n-3 LCPUFA group, compared to no change in the placebo group (P < 0.001). The ratio of DHA+EPA:arachidonic acid (AA) increased by 42 % in the n-3 LCPUFA group and by 5 % in the placebo group (P < 0.001). Bone-specific alkaline phosphatase and osteocalcin decreased in the n-3 LCPUFA group (P < 0.05) with no between-group difference. Short-term n-3 LCPUFA supplementation increased RBC concentrations of DHA and n-3:n-6 ratios. Bone turnover decreased with n-3 LCPUF, but not statistically compared to placebo. The results point to the need for investigations with greater dosages of n-3 LCPUFA for a longer duration to understand the contribution to bone metabolism in postmenopausal women.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0124-0132. DOI:10.1024/0300-9831/a000199
  • [Show abstract] [Hide abstract]
    ABSTRACT: This triple-blind randomized controlled clinical trial was conducted on 88 type 2 diabetic (T2DM) patients (males and females). Subjects in the fenugreek seed (n = 44) and placebo (n = 44) groups consumed 10 g/d of powdered whole fenugreek seeds or 5 g/d of wheat starch for 8 weeks. Fasting blood samples, anthropometric measurements and dietary records were collected at the baseline and at the end of the trial. Fenugreek seeds significantly decreased fasting blood glucose (P = 0.007) and HbA1c (P = 0.0001), serum levels of insulin (P = 0.03), homeostatic model assessment for insulin resistance (P = 0.004), total cholesterol (P = 0.005) and triglycerides (P = 0.0001) and increased serum levels of adiponectin (P = 0.001) compared with placebo. No significant changes were shown in serum low-density lipoprotein cholesterol and high-density lipoprotein cholesterol in both groups. In conclusion, fenugreek seeds improved glucose metabolism, serum lipid profile and adiponectin levels in studied subjects, and may be useful in the control of diabetes risk factors in TD2M patients.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0196-0205. DOI:10.1024/0300-9831/a000206
  • [Show abstract] [Hide abstract]
    ABSTRACT: Food supplements, if not properly used, may lead to potentially harmful nutrient intake. The purpose of this survey was to examine vitamin intake from food supplements. Taking into account the intake from food, as obtained from the National Nutrition Survey, it was determined whether the tolerable upper intake levels (ULs) were exceeded via supplements alone, or in combination with food. Data from 1070 supplement users (18 - 93 years) was available. The dietary and supplemental vitamin intakes of three groups were analyzed: average intake (50th percentile food + 50th percentile supplements), middle-high intake (50th + 95th ) and high intake (95th + 95th ). Vitamin C (53 %), vitamin E (45 %) and B vitamins (37 - 45 %) were consumed most frequently. Few subjects (n = 7) reached or exceeded the ULs through supplements alone. The UL for vitamin A and folate was reached by a few men in the middlehigh group, and by a few men and women in the high intake group. Otherwise, even in the high intake group, the recommended vitamin D intake of 20 μg/day (in case of insufficient endogenous synthesis) could not be achieved. The use of food supplements was not associated with excessive vitamin intake in this survey, except in a small number of cases. Vitamin A intake above the UL was the result of high dietary intake which also included the intake of β-carotene, rather than the result of overconsumption of food supplements. Diets mainly included folate from natural sources, which has no associated risk.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0152-0162. DOI:10.1024/0300-9831/a000202
  • [Show abstract] [Hide abstract]
    ABSTRACT: Glutamate is the major mediator of excitatory signals in the mammalian central nervous system. Extreme amounts of glutamate in the extracellular spaces can lead to numerous neurodegenerative diseases. We aimed to clarify the potential of the following vitamin E isomers, tocotrienol-rich fraction (TRF) and α-tocopherol (α-TCP), as potent neuroprotective agents against glutamate-induced injury in neuronal SK-N-SH cells. Cells were treated before and after glutamate injury (pre- and posttreatment, respectively) with 100 - 300 ng/ml TRF/α-TCP. Exposure to 120 mM glutamate significantly reduced cell viability to 76 % and 79 % in the pre- and post-treatment studies, respectively; however, pre- and post-treatment with TRF/α-TCP attenuated the cytotoxic effect of glutamate. Compared to the positive control (glutamate-injured cells not treated with TRF/α-TCP), pre-treatment with 100, 200, and 300 ng/ml TRF significantly improved cell viability following glutamate injury to 95.2 %, 95.0 %, and 95.6 %, respectively ( p < 0.05).The isomers not only conferred neuroprotection by enhancing mitochondrial activity and depleting free radical production, but also increased cell viability and recovery upon glutamate insult. Our results suggest that vitamin E has potent antioxidant potential for protecting against glutamate injury and recovering glutamate-injured neuronal cells. Our findings also indicate that both TRF and α-TCP could play key roles as anti-apoptotic agents with neuroprotective properties.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0140-0151. DOI:10.1024/0300-9831/a000201
  • [Show abstract] [Hide abstract]
    ABSTRACT: Background: The prevalence of diabetes is growing worldwide. The primary symptom of diabetes mellitus is elevated blood sugar. This is usually treated with lifestyle intervention and drugs according to an algorithm based on glycated hemoglobin (HbA1c) levels. We present the case of a patient who successfully managed his type 2 diabetes solely through lifestyle modification. Case Report: A 45-year-old businessman with a body mass index of 27 kg/m2 was examined within a secondary prevention program in Austria. His HbA1c was 9.7 % - type 2 diabetes mellitus was diagnosed. General recommendations for lifestyle were given and metformin was prescribed. Upon his diagnosis the patient searched for all the information he could get about diabetes and implemented this new knowledge in his everyday life. He had a strong desire to defeat his disease and he wanted to stop using medications. He identified some nutritional ingredients and spices that affected his blood sugar in a positive way. He stopped taking metformin after 4 weeks and handled his diabetes with his personal lifestyle program. Three months after the diagnosis his HbA1c was 6.4 %; after 6 months he had an HbA1c of 6.0 % without the use of medication. Discussion: Usually, multiple drug therapy is necessary to handle high blood glucose levels. Our business manager ate as much as before his diagnosis but he modified the contents of his diet so that the lifestyle intervention was not hard for him. General recommendations for lifestyle modification usually include: more exercise, reduced sugar and monosaccharides, and less alcohol and nicotine. With the knowledge of the effects of specific dietary ingredients, it might be possible to modify a regular diet in such a way as to benefit people with type 2 diabetes, to substantially improve quality of life.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0133-0139. DOI:10.1024/0300-9831/a000200
  • [Show abstract] [Hide abstract]
    ABSTRACT: Findings of studies on the link between dietary acid-base balance and bone mass are relatively mixed. We examined the association between dietary acid-base balance and bone mineral density (BMD) in a sample of Iranian women, hypothesizing that a higher dietary acidity would be inversely associated with BMD, even when dietary calcium intake is adequate. In this cross-sectional study, lumbar spine and femoral neck BMDs of 151 postmenopausal women aged 50 - 85 years were measured using dual-energy x-ray absorptiometry. Dietary intakes were assessed using a validated food frequency questionnaire. Renal net acid excretion (RNAE), an estimate of acid-base balance, was then calculated indirectly from the diet using the formulae of Remer (based on dietary intakes of protein, phosphorus, potassium, and magnesium; RNAE Remer ) and Frassetto (based on dietary intakes of protein and potassium; RNAE Frassetto ), and was energy adjusted by the residual method. After adjusting for potential confounders, multivariable adjusted means of the lumbar spine BMD of women in the highest tertiles of RNAE Remer and RNAE Frassetto were significantly lower than those in the lowest tertiles (for RNAE Remer: mean difference -0.084 g/cm2 ; P =0.007 and for RNAE Frassetto: mean difference - 0.088 g/cm2 ; P =0.004). Similar results were observed in a subgroup analysis of subjects with dietary calcium intake of >800 mg/ day. In conclusion, a higher RNAE (i. e. more dietary acidity), which is associated with greater intake of acid-generating foods and lower intake of alkali-generating foods, may be involved in deteriorating the bone health of postmenopausal Iranian women, even in the context of adequate dietary calcium intake.
    International Journal for Vitamin and Nutrition Research 05/2015; 84(3-4):0206-0217. DOI:10.1024/0300-9831/a000207

  • International Journal for Vitamin and Nutrition Research 01/2015;
  • Source
    [Show abstract] [Hide abstract]
    ABSTRACT: Isotope dilution methods have been successfully used to estimate vitamin A status in human populations as well as to evaluate the impact of vitamin A interventions. The most commonly applied isotope dilution method is the retinol isotope dilution technique, which is based on the 1989 “Olson equation” for estimating total body vitamin A stores (sometimes equated to liver vitamin A) after an oral dose of labeled vitamin A. The equation relies on several factors related to absorption and retention of the dose, the equilibration of label in plasma vs. liver, and timing of a blood sample for measurement of labeled vitamin A. Here, the assumptions underlying these factors are discussed, and new results based on applying model-based compartmental analysis [specifically, the Simulation, Analysis and Modeling software (WinSAAM)] to data on retinol kinetics in humans are summarized. A simplification of the Olson equation, in which plasma tracer is measured 3 days after administration of the oral dose and several factors are eliminated, is presented. The potential usefulness of the retinol isotope dilution technique for setting vitamin A requirements and assessing vitamin A status in children, as well as the confounding effects of inflammation and likely variability in vitamin A absorption, are also discussed.
    International Journal for Vitamin and Nutrition Research 12/2014; 84(Supplement 1):9-15. DOI:10.1024/0300-9831/a000181
  • Source

    International Journal for Vitamin and Nutrition Research 12/2014; 84(Supplement 1):7-8. DOI:10.1024/0300-9831/a000180
  • Source
    [Show abstract] [Hide abstract]
    ABSTRACT: A variety of methods exist to assess vitamin A status of groups and populations. Vitamin A status is usually defined by the liver retinol concentration. Most indicators of status do not measure or estimate liver stores of retinol. Clinical signs only have utility when liver reserves are almost exhausted, and serum retinol concentrations have utility in the zone of overt deficiency. Dose response tests offer more coverage, but cannot distinguish among liver vitamin A stores in the adequate through toxic range. Different countries continue, or are beginning, to add preformed vitamin A to a variety of staple foods through fortification, and vitamin A supplements are still being distributed in many countries, especially to preschool children. Further, provitamin A biofortified crops are currently being released in several countries. Assessing population vitamin A status in response to these interventions needs to move beyond serum retinol concentrations. Indicators that work in the excessive to toxic range of liver reserves are needed. To date, the only indirect indicator that has been validated in this range of liver reserves in animals and humans is the retinol isotope dilution test using deuterium or 13C, which spans the entire liver reserve continuum from deficiency through excess.
    International Journal for Vitamin and Nutrition Research 12/2014; 84(Supplement 1):16-24. DOI:10.1024/0300-9831/a000182