Journal of Pediatric Gastroenterology and Nutrition Impact Factor & Information

Publisher: North American Society for Pediatric Gastroenterology and Nutrition; European Society for Paediatric Gastroenterology and Nutrition; North American Society for Pediatric Gastroenterology, Hepatology and Nutrition; European Society for Pediatric Gastroenterology, Hepatology and Nutrition, Lippincott, Williams & Wilkins

Journal description

In addition to providing up-to-date information, the Journal of Pediatric Gastroenterology and Nutrition has been developed with the intention of promoting and enhancing communication throughout the gastroenterology community.

Current impact factor: 2.87

Impact Factor Rankings

2015 Impact Factor Available summer 2015
2013 / 2014 Impact Factor 2.873
2012 Impact Factor 2.196
2011 Impact Factor 2.298
2010 Impact Factor 2.18
2009 Impact Factor 2.183
2008 Impact Factor 2.132
2007 Impact Factor 2.102
2006 Impact Factor 2.067
2005 Impact Factor 2.077
2004 Impact Factor 1.764
2003 Impact Factor 1.402
2002 Impact Factor 2.078
2001 Impact Factor 2.077
2000 Impact Factor 1.58
1999 Impact Factor 1.486
1998 Impact Factor 1.319
1997 Impact Factor 1.294
1996 Impact Factor 1.523
1995 Impact Factor 1.243
1994 Impact Factor 1.082
1993 Impact Factor 0.925
1992 Impact Factor 0.893

Impact factor over time

Impact factor
Year

Additional details

5-year impact 2.38
Cited half-life 7.20
Immediacy index 0.79
Eigenfactor 0.01
Article influence 0.66
Website Journal of Pediatric Gastroenterology and Nutrition website
Other titles Journal of pediatric gastroenterology and nutrition, JPGN, J pediatr gastroenterol nutr
ISSN 0277-2116
OCLC 7520467
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details

Lippincott, Williams & Wilkins

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • Pre-print must be removed upon acceptance for publication
    • Post-print may be deposited in personal website or institutional repository
    • Publisher's version/PDF cannot be used
    • Must include statement that it is not the final published version
    • Published source must be acknowledged with full citation
    • Set statement to accompany deposit
    • Must link to publisher version
    • NIH authors will have their accepted manuscripts transmitted to PubMed Central on their behalf after a 12 months embargo (see policy for details)
    • Wellcome Trust and HHMI authors will have their accepted manuscripts transmitted to PubMed Central on their behalf after a 6 months embargo (see policy for details)
    • Publisher last reviewed on 19/03/2015
  • Classification
    ​ yellow

Publications in this journal

  • Journal of Pediatric Gastroenterology and Nutrition 04/2015; DOI:10.1097/MPG.0000000000000798
  • Journal of Pediatric Gastroenterology and Nutrition 04/2015;
  • Journal of Pediatric Gastroenterology and Nutrition 03/2015; 60(3):289-90. DOI:10.1097/MPG.0000000000000603
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    ABSTRACT: The North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition has formulated guidelines for managing functional constipation. There have been no studies that have investigated how pediatricians apply the constipation guideline since it was revised in 2006. The purpose of this study was to examine how pediatricians approach functional constipation and how closely their approaches adhere to the guidelines. An anonymous multiple-choice questionnaire was developed by general pediatricians and pediatric gastroenterologists. This was distributed to pediatricians and pediatric residents at 7 academic institutions, and to the American Academy of Pediatrics section on medical students, residents, and fellowship trainees mailing list. A total of 1202 responses were received (952 trainees, 250 attendings). Of these, 84.3% reported being unfamiliar/slightly familiar with the guidelines. The most common initial interventions for constipation without fecal incontinence included fluids (92.1%), fiber (89.5%), juice (77.7%), behavioral interventions (71.2%), follow-up (53.4%), and reducing constipating foods (50.1%). The most common initial interventions for constipation with fecal incontinence included bowel cleanout (73.4%), maintenance medication (70.0%), fluids (67.9%), behavioral interventions (67.6%), fiber (66.1%), and follow-up (57.8%). Osmotics were the most commonly prescribed as needed (83.0%) and maintenance medications (96.8%), with stimulants prescribed PRN by 35.6% and as maintenance by 16.8%. Some individuals (39.7%) reported concern that osmotics could result in dependence, addiction, or electrolyte imbalances, compared with 73.0% for stimulants. Our results show that more education regarding medication in functional constipation is necessary, including the use of medication reducing time to remission, the necessity of disimpaction, and misconceptions regarding adverse effects.
    Journal of Pediatric Gastroenterology and Nutrition 03/2015; 60(3):308-11. DOI:10.1097/MPG.0000000000000591
  • Journal of Pediatric Gastroenterology and Nutrition 03/2015; 60(3):291-2. DOI:10.1097/MPG.0000000000000655
  • Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000772
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    ABSTRACT: The ability of the World Health Organization (WHO) growth standards to represent the growth of South East Asian infants has been questioned. The aim of this study was to provide contemporary data on the growth of Indonesian breast-fed and formula-fed infants, compared with the WHO growth standards. A prospective cohort study of 160 normal healthy infants was undertaken in a suburban area of South Jakarta, Indonesia. Infants from 2-6 weeks of age were recruited, and consumed exclusively either breast milk or infant formula for at least 6 months, with follow-up until 12 months of age. Overall, the infants in this study were lighter (weight-for-age), were shorter (length-for-age) and had smaller head circumferences (head circumference-for-age) than the average WHO Growth Reference Study (WGRS) population but were of similar proportion (weight-for-length). Compared with the WGRS, the z-scores for weight-for-age, length-for-age and head circumference-for-age in the Indonesian children fell from birth to 6 weeks of age and then increased until 3 months of age in both the breast-fed infants and the formula-fed infants. At 6 weeks of age, the weight-for-age z-scores fell below -2 standard deviations (SD) for 16 (20.5%) breast-fed and 40 (51.3%) formula-fed infants, and the length-for-age z-scores fell below -2 SD for 31 (39.7%) breast-fed and 41 (52.6%) formula-fed infants. The WHO growth standards do not reflect the growth of this cohort of Indonesian infants and may overestimate the levels of underweight and stunted children. ClinicalTrials.gov Identifier: NCT01721512This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial No-Derivatives 4.0 License, where it is permissible to download and share the work, provided it is properly cited. The work cannot be changed in any way or used commercially. http://creativecommons.org/licenses/by-nc-nd/4.0.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000770
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    ABSTRACT: While reduced cognitive function has been demonstrated after liver transplantation in children, less data are available concerning motor competence. Thirty-five children aged 4-12 years were tested using Movement Assessment Battery for Children (M-ABC) test at a median of 5.1(3.9-6.9) years after liver transplantation and compared to reference material of healthy children. Transplanted children had worse M-ABC score 8.0(interquartile range 5.0-11.5), compared to healthy children 3.5(1.0-6.0)(P < 0.0001). All sub-scores (manual dexterity (P < 0.0001), ball skills (P = 0.0037), balance (P = 0.0032)) were significantly worse in the liver transplanted children compared to the healthy reference group. 29% of the liver transplanted children had impaired motor competence, compared to 9% of a healthy reference group. Seventeen of the transplanted patients were retested one year later, and eleven were tested 4 years later with no changes in total M-ABC score. Ball skill competence was worse 4 years after first assessment (P = 0.013). For transplanted children with cholestatic liver disease (n = 26) renal function was a significant predictor for total M-ABC score (P = 0.018). Liver transplanted children had impaired motor competence compared to healthy children. Ball skills developed adversely several years after liver transplantation and motor competence did not improve with time after transplantation. Renal function was a significant predictor for motor competence in liver transplanted children with cholestatic liver disease.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000757
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    ABSTRACT: Biliary atresia is a rare disease of unclear etiology, where obstruction of the biliary tree causes severe cholestasis leading to cirrhosis and ultimate death if left untreated. Biliary atresia is the leading cause of neonatal cholestasis and the most frequent indication for pediatric liver transplantation. Any infant with persistent jaundice beyond 2 weeks of life needs to be evaluated for biliary atresia with fractionation of the bilirubin into a conjugated and unconjugated portion. Early performance of a hepatoportoenterostomy in the first 45 days of life to restore bile flow and lessen further damage to the liver is thought to optimize outcome. Despite surgery, progressive liver scarring occurs and 80% of patients with biliary atresia will require liver transplantation during childhood.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000755
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    ABSTRACT: Identification of patients at risk for developing cirrhotic cystic fibrosis liver disease (CCFLD) is essential for targeting potentially preventive treatment. We studied the evolution of serum liver enzymes and thrombocyte counts as predictors of CCFLD development. For this study, we defined the diagnosis of CCFLD as the combination of splenomegaly (on either physical examination or ultrasound scan) and macronodularity of the liver on ultrasound scan. We reviewed the medical records of 277 pediatric CF patients for the diagnosis of CCFLD. In each CCFLD patient, we reviewed serum liver enzymes and thrombocyte counts in the two years period preceding the diagnosis of CCFLD. We compared these results with a non-CCFLD control group (CF patients over 15 years of age without any reported signs or symptoms of CCFLD). In the two years preceding the diagnosis, the GGT levels of CCFLD patients were significantly higher compared to non-CCFLD controls (42 ± 5 vs.17 ± 2 U/L, respectively, P < 0.001). Corresponding AST and ALT levels did not significantly differ between CCFLD and control patients. The thrombocyte counts in CCFLD patients were significantly lower than those in controls (252 ± 108 vs. 320 ± 94 × 10/l, respectively, P < 0.05). The predictive value, for CCFLD, of a single GGT measurement was low. However, for CF patients with a mean GGT>35 U/L, based on repeated measurements, the Odds ratio for developing CCFLD was 39 (95% CI: [9-175], specificity: 95% sensitivity: 64%, positive predictive value: 50%). For the thrombocytes, however, no reliable cutoff value could be identified. In pediatric CF patients a, persistently, high-normal GGT is strongly associated with the diagnosis of CCFLD within two years. The prognostic value of a single GGT measurement is limited, but repeated GGT measurements may allow to identify (groups of) patients at increased risk for CCFLD.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000758
  • Journal of Pediatric Gastroenterology and Nutrition 02/2015; 60(4). DOI:10.1097/MPG.0000000000000756
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    ABSTRACT: Our aim was to evaluate whether a subset of non-critically ill convalescing ELBW neonates who were managed using a standardized nutritional protocol could achieve the growth rate of the index fetus. We hypothesized that an aggressive nutritional protocol applied to non-critically ill, convalescing premature neonates could achieve the growth rate of the index fetus. This was a retrospective review of a subset of 21 premature neonates defined by four criteria: i) inborn or transferred-in within 24 hours of birth, ii) gestational age (GA) ≤ 30 weeks and birth weight (BW) ≤1000 g, iii) hospitalized >35 postnatal days and iv) discharged between 34-42 weeks post-conceptual age. Optimal growth at discharge was defined as weight and head circumference (HC) >10 percentile compared to comparable GA fetal parameters. Protein intake of ≥1.4 g/Kg/d and energy ≥ 30kcal/Kg/d was provided as of the first postnatal day. Protein ≥3 g/Kg/d and >80 kcal/Kg/d were established ≥10 postnatal day. Birth weight was regained by postnatal day 10 ± 5 d; (mean + SD). Nutrition was predominantly enteral (i.e. > 50% of all calories) after the 11 postnatal day. At discharge, 71% (15/21) by weight and 76% (16/21) by HC were >10 percentile. After 30 week post-conceptual age, the cohort exceeded the weight gain rate (g/week) of a 10th percentile fetus. In a selected subset of non-critically ill convalescing ELBW neonates, after a brief period of unavoidable postnatal weight loss and body water adjustment, adequate nutrition from birth can enable a more homeostatic pattern of growth that approximates growth of the index fetus.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000750
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    ABSTRACT: This study tested the hypothesis that the fecal bacterial genera of breast-fed (BF) and formula-fed (FF) infants differ and that human milk oligosaccharides (HMO) modulate the microbiota of BF infants. Fecal samples were obtained from BF (n = 16) or FF (n = 6) infants at 3-month postpartum. Human milk were collected on the same day when feces were collected. The microbiota was assessed by pyrosequencing of bacterial 16S rRNA genes. HMO were measured by HPLC-Chip time-of-flight mass spectrometry. The overall microbiota of BF differed from that of FF (P = 0.005). Compared to FF, BF had higher relative abundances of Bacteroides, lower proportions of Clostridium XVIII, Lachnospiracea incertae sedis, Streptococcus, Enterococcus and Veillonella (P < 0.05). Bifidobacterium predominated in both BF and FF infants, with no difference in abundance between the two groups. The most abundant HMO were lacto-N-tetraose + lacto-N-neotetraose (LNT + LNnT, 22.6%), followed by 2'-fucosyllactose (2'FL, 14.5%) and lacto-N-fucopentaose I (LNFP I, 9.5%). Partial least squares regression of HMO and microbiota showed several infant fecal bacterial genera could be predicted by their mothers' HMO profiles and the important HMO for the prediction of bacterial genera were identified by variable importance in the projection scores. These results strengthen the established relationship between HMO and the infant microbiota, identify statistical means whereby infant bacterial genera can be predicted by milk HMO. Future studies are needed to validate these findings and determine if supplementation of formula with defined HMO could selectively modify the gut microbiota.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000752
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    ABSTRACT: Oral high dose repletion vitamin D therapy, also known as stoss therapy, can be effective in the treatment of nutritional vitamin D-deficiency rickets in infants and young children without liver disease and in patients with cystic fibrosis. There is no literature about this approach in infants with new onset cholestasis. This was a retrospective chart review of cholestatic infants from March 2010 to March 2012 at a pediatric tertiary care center. Four cases satisfied the inclusion criteria and were described in detail. All patients received oral high dose repletion therapy with ergocalciferol (vitamin D2) 300,000 IU daily for 2 to 3 days. Follow-up vitamin D levels approximately 4 weeks later showed failure to achieve sufficiency levels (> 20 ng/dl) in any patient. Unlike infants without liver disease, use of oral high dose repletion therapy may not be adequate as treatment of vitamin D deficiency in the setting of cholestasis.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; DOI:10.1097/MPG.0000000000000751
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    ABSTRACT: Adverse early life events are key factors for development of functional gastrointestinal disorders (FGIDs). Urinary tract infection (UTI) is associated with chronic pelvic pain in adults, a finding that has been recapitulated in murine models, but the relation between UTI and chronic pelvic and abdominal pain has not been studied in children. We hypothesized that UTI in infancy increases the risk of FGIDs and chronic abdominal pain (CAP) in childhood. The present study included children, ages 4 to 18 years, with a single UTI in the first year of life and their siblings with no history of UTI. Parents completed the Questionnaire on Pediatric Gastrointestinal Symptoms-Rome III Version (QPGS-III) by telephone. Children meeting QPGS-III criteria for FGIDs but with pain less than once weekly were considered to have CAP. A total of 57 patients with UTI and 58 sibling controls were identified. Mean age at UTI was 4.8 months, and mean time since UTI was 9.3 years. At the time of survey, mean age of patients was 9.7 years (5-16 years, 40% boys) and that of controls was 9.6 years (range 4-17 years, 57% boys). FGIDs were diagnosed in 6 of 57 (11%) patients, and 1 of 58 (2%) controls (P = 0.06). CAP was identified in 10 of 57 (18%) patients and 2 of 58 (3%) controls (P = 0.02). Predominant sex (female), infecting organism (E coli), and treatment (third-generation cephalosporin) were similar in patients with UTI with and without CAP. We show for the first time that UTI is associated with CAP in childhood. We speculate that pelvic organ sensory convergence explains our findings.
    Journal of Pediatric Gastroenterology and Nutrition 02/2015; 60(2):214-6. DOI:10.1097/MPG.0000000000000599