Surgical Neurology (SURG NEUROL )
Surgical Neurology provides outstanding, comprehensive coverage of the latest developments in the field of neurosurgery by providing peer-reviewed articles that discuss advances in clinical practice and research of value for the practicing neurosurgeon and resident. In 1995, Surgical Neurology broadened its editorial scope to include information for the practicing neurosurgeon-in and out of the OR: the journal now features controversial editorials on scientific and non-scientific issues that concern neurosurgeons in today's changing world, including, health care, malpractice, practice building and development, advice to young neurosurgeons, and retirement.
Impact factor 1.67
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- 5-year impact1.86
- Cited half-life0.00
- Immediacy index0.22
- Article influence0.58
- WebsiteSurgical Neurology website
- Other titlesSurgical neurology
- Material typePeriodical, Internet resource
- Document typeJournal / Magazine / Newspaper, Internet Resource
- Author can archive a pre-print version
- Author can archive a post-print version
- Pre-print allowed on any website or open access repository
- Voluntary deposit by author of authors post-print allowed on authors' personal website, arXiv.org or institutions open scholarly website including Institutional Repository, without embargo, where there is not a policy or mandate
- Deposit due to Funding Body, Institutional and Governmental policy or mandate only allowed where separate agreement between repository and the publisher exists.
- Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months .
- Set statement to accompany deposit
- Published source must be acknowledged
- Must link to journal home page or articles' DOI
- Publisher's version/PDF cannot be used
- Articles in some journals can be made Open Access on payment of additional charge
- NIH Authors articles will be submitted to PubMed Central after 12 months
- Publisher last contacted on 18/10/2013
- Classification green
Publications in this journal
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ABSTRACT: Abstract Background: Fusiform intracranial aneurysms (FIAs) are uncommon lesions representing less than 15% of all intracranial aneurysms in most large series. Their etiology has been linked to a variety of causes including atherosclerosis, fibromuscular dysplasia, cystic medial necrosis, connective tissue disease, hypertension, diabetes, hyperlipidemia, infection, cardiac myxoma, oral contraceptive use, vasculitis, and lymphoproliferative disorders. The finding of numerous lesions in a single patient is distinctly uncommon. Case Description: We describe the unique case of a 47‑year‑old female who developed multiple FIAs over a 6‑year period without an obvious underlying pathology. The patient’s medical history was significant for obesity, migraine headaches, insomnia, breast cancer, and chronic skin rash. Various diagnoses were explored including infectious etiologies, autoimmune vasculopathies, malignancy‑related causes, connective tissue disorders, and underlying genetic conditions. However, all investigations, including aneurysm wall and skin biopsies were negative or deemed noncontributory toward making a definitive diagnosis. Conclusion: We report an unusual case of a patient with a normal cerebral angiogram developing numerous, FIAs without obvious underlying etiology over a 6‑year period. Close clinical and radiological follow‑up is recommended in this case because the natural history of the disease is unclear at this point. The literature regarding potential causes of multiple fusiform intracranial aneuryms is reviewed.Surgical Neurology 07/2014; 5(107).
- Surgical Neurology 11/2009; 72(5):526.
- Surgical Neurology 11/2009; 72(5):522.
- Surgical Neurology 11/2009; 72(5):529.
- Surgical Neurology 11/2009; 72(5):516.
- Surgical Neurology 07/2009; 71(6):638-9.
- Surgical Neurology 07/2009; 71(6):725-6.
- Surgical Neurology 06/2009; 71(5):526.
Article: The lesson of anatomy.Surgical Neurology 06/2009; 71(5):597-8; discussion 598-9.
- Surgical Neurology 06/2009; 71(5):525.
Article: Research news and notes.Surgical Neurology 06/2009; 71(5):523-524.
Article: Commentary.Surgical Neurology 06/2009; 71(5):598-599.
Article: In this issue...Surgical Neurology 06/2009; 71(6):627-8.
Article: "Hanging until death?".Surgical Neurology 05/2009; 71(4):518.
Article: Crisis! Crisis! Crisis?Surgical Neurology 05/2009; 71(4):516-7.
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ABSTRACT: BackgroundRisks related to rebleeding of a ruptured intracranial aneurysm have decreased. However, ischemic neurologic deficits related to vasospasm are still the leading causes of mortality and morbidity. It is well known that vasospasm is a dynamic process affected by various factors. The severity of vasospasm in animal models and clinical observations differ from each other. This variability has not been completely explained by blood and blood degradation products. Therefore, metabolites released from the damaged vessel wall during the bleeding are thought to play an important role in vasospasm.MethodTo test this hypothesis, we used 46 male Wistar rats that were divided into 7 groups and administered one of the following to cisterna magna: venous blood, arterial blood, arterial wall homogenate, venous wall homogenate, combined mixture of arterial blood and artery wall homogenate, or combined mixture of venous blood and venous wall homogenate. Brainstems of the rats were excised, and the basilar arteries were harvested for morphometric measurements.ResultThere were significant differences between the degree of vasospasm caused by arterial and venous blood (P < .0001). The intraluminal area of the basilar artery was significantly narrower after application of arterial blood, artery wall homogenate, or their combination (49% ± 1%) than after venous groups (30% ± 1.9%) (P < .0001).ConclusionThe results of this experiment demonstrated that metabolites from vessel walls play as important roles in the pathophysiology of vasospasm as blood and blood degradation products. Further investigation of these metabolites will improve our understanding of vasospasm, pathophysiology, and its treatment.Surgical Neurology 05/2009;
Article: Commentary.Surgical Neurology 05/2009; 71(4):418.
Data provided are for informational purposes only. Although carefully collected, accuracy cannot be guaranteed. The impact factor represents a rough estimation of the journal's impact factor and does not reflect the actual current impact factor. Publisher conditions are provided by RoMEO. Differing provisions from the publisher's actual policy or licence agreement may be applicable.