Journal of Child Psychology and Psychiatry (J CHILD PSYCHOL PSYC)

Publisher: Association for Child Psychology and Psychiatry, Wiley

Journal description

The Journal of Child Psychology and Psychiatry is internationally recognised to be the leading journal covering both child and adolescent psychology and psychiatry. Articles published include experimental and developmental studies, especially those relating to developmental psychopathology and the developmental disorders. An important function of the Journal is to bring together empirical research, clinical studies and reviews of high quality arising from different theoretical perspectives.

Current impact factor: 5.67

Impact Factor Rankings

2015 Impact Factor Available summer 2015
2013 / 2014 Impact Factor 5.669
2012 Impact Factor 5.422
2011 Impact Factor 4.281
2010 Impact Factor 4.36
2009 Impact Factor 4.983
2008 Impact Factor 4.854
2007 Impact Factor 4.432

Impact factor over time

Impact factor
Year

Additional details

5-year impact 6.24
Cited half-life 8.50
Immediacy index 0.98
Eigenfactor 0.03
Article influence 2.30
Website Journal of Child Psychology and Psychiatry and Allied Disciplines, The website
Other titles Journal of child psychology and psychiatry and allied disciplines, Journal of child psychology and psychiatry
ISSN 0021-9630
OCLC 1307942
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details

Wiley

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 2 years embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • On author's personal website, institutional repositories, arXiv, AgEcon, PhilPapers, PubMed Central, RePEc or Social Science Research Network
    • Author's pre-print may not be updated with Publisher's Version/PDF
    • Author's pre-print must acknowledge acceptance for publication
    • On a non-profit server
    • Publisher's version/PDF cannot be used
    • Publisher source must be acknowledged with citation
    • Must link to publisher version with set statement (see policy)
    • If OnlineOpen is available, BBSRC, EPSRC, MRC, NERC and STFC authors, may self-archive after 12 months
    • If OnlineOpen is available, AHRC and ESRC authors, may self-archive after 24 months
    • Publisher last contacted on 07/08/2014
    • This policy is an exception to the default policies of 'Wiley'
  • Classification
    ​ yellow

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Background: ADHD-like symptoms are common in FASD. FASD and ADHD groups both display executive function impairments, however, there is ongoing debate whether the pattern and magnitude of executive function deficits differs between these two types of disorders. Methods: An electronic literature search was conducted (PubMed, PsychInfo; 1972-2013) to identify studies comparing the executive functioning of children with FASD with ADHD or control groups. FASD groups included those with and without dysmorphy (i.e., FAS, pFAS, ARND, and other FASD diagnoses). Effect sizes (Hedges’ g, standardized mean difference) were calculated. Random effects meta-analytic models were performed using the metafor package for R. Results: Fifty-one studies met inclusion criteria (FASD N=2,115; ADHD N=453; controls N=1,990). Children with FASD showed the strongest and most consistent deficits in planning, fluency, and set-shifting compared to controls (Hedges’ g=-0.94, -0.78) and children with ADHD (Hedges’ g=-0.72, -0.32). FASD was associated with moderate to large impairments in working memory, compared to controls (Hedges’ g= -.84, -.58) and small impairments relative to groups with ADHD (Hedges’ g= -.26). Smaller and less consistent deficits were found on measures of inhibition and vigilance relative to controls (Hedges’ g=-0.52, -0.31); FASD and ADHD were not differentiated on these measures. Moderator analyses indicated executive dysfunction was associated with older age, dysmorphy, and larger group differences in IQ. Sex and diagnostic system were not consistently related to effect size. Conclusions: While FASD is associated with global executive impairments, executive function weaknesses are most consistent for measures of planning, fluency, and set-shifting. Neuropsychological measures assessing these executive function domains may improve differential diagnosis and treatment of FASD.
    Journal of Child Psychology and Psychiatry 06/2015; In Press.
  • Journal of Child Psychology and Psychiatry 03/2015;
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    ABSTRACT: This study examined whether relocating from a high-poverty neighborhood to a lower poverty neighborhood as part of a federal housing relocation program (HOPE VI; Housing Opportunities for People Everywhere) had effects on adolescent mental and behavioral health compared to adolescents consistently living in lower poverty neighborhoods. Sociodemographic, risk behavior, and neighborhood data were collected from 592 low-income, primarily African-American adolescents and their primary caregivers. Structured psychiatric interviews were conducted with adolescents. Prerelocation neighborhood, demographic, and risk behavior data were also included. Hierarchical Linear Modeling (HLM) was used to test associations between neighborhood variables and risk outcomes. HLM was used to test whether the effect of neighborhood relocation and neighborhood characteristics might explain differences in sexual risk taking, substance use, and mental health outcomes. Adolescents who relocated of HOPE VI neighborhoods (n = 158) fared worse than control group participants (n = 429) on most self-reported mental health outcomes. The addition of subjective neighborhood measures generally did not substantively change these results. Our findings suggest that moving from a high-poverty neighborhood to a somewhat lower poverty neighborhood is not associated with better mental health and risk behavior outcomes in adolescents. The continued effects of having grown up in a high-poverty neighborhood, the small improvements in their new neighborhoods, the comparatively short length of time they lived in their new neighborhood, and/or the stress of moving appears to worsen most of the mental health outcomes of HOPE VI compared to control group participants who consistently lived in the lower poverty neighborhoods. © 2015 Association for Child and Adolescent Mental Health.
    Journal of Child Psychology and Psychiatry 02/2015; DOI:10.1111/jcpp.12386
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    ABSTRACT: We examined whether the temperament of behavioral inhibition is a significant marker for psychopathology in early adulthood and whether such risk is buffered by peer social networks. Participants (N = 165) were from a prospective study spanning the first two decades of life. Temperament was characterized during infancy and early childhood. Extent of involvement in peer social networks was measured during adolescence, and psychopathology was assessed in early adulthood. Latent Class Analyses generated comprehensive variables at each of three study time-points. Regressions assessed (a) the direct effect of early behavioral inhibition on adult psychopathology (b) the moderating effect of adolescent involvement in social peer networks on the link between temperamental risk and adult psychopathology. Stable behavioral inhibition in early childhood was negatively associated with adult mental health (R(2 ) = .07, p = .005, β = -.26), specifically increasing risk for adult anxiety disorders (R(2) = .04, p = .037, β = .19). These temperament-pathology relations were significantly moderated by adolescent peer group social involvement and network size (Total R(2) = .13, p = .027, β = -.22). Temperament predicted heightened risk for adult anxiety when adolescent social involvement was low (p = .002, β = .43), but not when adolescent social involvement was high. Stable behavioral inhibition throughout early childhood is a risk factor for adult anxiety disorders and interacts with adolescent social involvement to moderate risk. This is the first study to demonstrate the critical role of adolescent involvement in socially active networks in moderating long-lasting temperamental risk over the course of two decades, thus informing prevention/intervention approaches. © 2015 Association for Child and Adolescent Mental Health.
    Journal of Child Psychology and Psychiatry 02/2015; DOI:10.1111/jcpp.12390
  • Journal of Child Psychology and Psychiatry 12/2014; 55(12):e6-e6. DOI:10.1111/jcpp.12361
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    ABSTRACT: Although long-term treatment is a core aspect of the management of children and adolescents with bipolar disorder (BD), most clinical recommendations are based on results from short-term studies or adult data. In order to guide clinical practice, we review the efficacy and safety profile of mood stabilizers, antipsychotics, and other pharmacological strategies for the long-term treatment of BD in pediatric patients.
    Journal of Child Psychology and Psychiatry 06/2014; 55(9). DOI:10.1111/jcpp.12271
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    ABSTRACT: Heterogeneity in the presentation, antecedents, prognosis and treatment response of antisocial behaviour has long provided a challenge to developmental psychopathology researchers. As illustrated in the incisive Frick and colleagues' Annual Research Review, there is growing evidence that the presence of high callous‐unemotional (CU) traits identifies a subgroup of antisocial young people with a particularly aggressive and pervasive form of disorder. Frick and colleagues extend their developmental psychopathology approach to CU traits by linking in theories of conscience development and considering evidence on the stability of CU traits. This commentary addresses these themes and the area more generally, considering (1) comparison of a CU specifier to alternative approaches to antisocial heterogeneity (2) high CU traits in the absence of antisocial behaviour and (3) aspects of the measurement of CU traits.
    Journal of Child Psychology and Psychiatry 06/2014; 55(6). DOI:10.1111/jcpp.12253
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    ABSTRACT: The field of developmental cognitive neuroscience is now established as a discipline at the nexus of the broader fields of developmental psychology and cognitive neuroscience. Sitting in its rear view mirror, but gaining rapidly, is the nascent discipline of developmental social neuroscience. Given the relative youth of this field, it is not surprising that a great deal of energy has gone into generating a rich corpus of empirical data.
    Journal of Child Psychology and Psychiatry 06/2014; 55(6). DOI:10.1111/jcpp.12254
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    ABSTRACT: We all have a tendency to be easily seduced by Occam's razor, or the desire to accept the simplest hypothesis to explain a given phenomenon. If we observe that children with ADHD come from more impoverished backgrounds or have diets that contain more additives than their peers, then a parsimonious theory would be that poverty and/or diet are causal factors in the development of ADHD. Such theories are all the more attractive because they suggest potential targets for intervention: improve family circumstances and child diet and symptoms of ADHD should diminish. Of course in reality untangling causal relationships is much more complicated. We now accept that many common disorders reflect the confluence of genetic and environmental risk factors, but that any given risk factor is likely to account for only a tiny amount of explained variance in symptom profile. What's more, in order to fulfil the promise of intervention, we must begin to unravel the precise mechanisms by which identified risk factors affect the developing system. This issue showcases two key methodologies for understanding causal influences on developmental disorders: longitudinal designs and well-controlled intervention studies. Both may employ statistical techniques that can identify the mediators of observed associations, elucidating potential mechanistic processes.
    Journal of Child Psychology and Psychiatry 05/2014; 55(5):413-415. DOI:10.1111/jcpp.12239
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    ABSTRACT: Background The relationship between inadequate sleep and mood has been well-established in adults and is supported primarily by correlational data in younger populations. Given that adolescents often experience shortened sleep on school nights, we sought to better understand the effect of experimentally induced chronic sleep restriction on adolescents' mood and mood regulation. Methods Fifty healthy adolescents, ages 14-17, completed a 3-week sleep manipulation protocol involving a baseline week, followed by a sleep restriction (SR) condition (6.5hr in bed per night for five nights) and healthy sleep duration (HS) condition (10hr in bed per night for five nights). The study used a randomized, counterbalanced, crossover experimental design. Participants' sleep was monitored at home via self-report and actigraphy. At the end of each condition, participants and their parents completed questionnaires of mood and mood regulation. To assess for expectancy effects, we also analyzed parent and teen ratings of hyperactivity/impulsivity, which prior research suggests is not sensitive to SR in adolescents. Wilcoxon Signed Rank tests compared questionnaire outcomes across the two conditions. ResultsParticipants averaged 2.5 more hours of sleep per night during HS relative to SR. Compared with HS, adolescents rated themselves as significantly more tense/anxious, angry/hostile, confused, and fatigued, and as less vigorous (p=.001-.01) during SR. Parents and adolescents also reported greater oppositionality/irritability and poorer emotional regulation during SR compared with HS (p<.05). There were no cross-condition differences in depression or hyperactivity/impulsivity (p>.05). Conclusions Findings complement prior correlational study results to show that after only a few days of shortened sleep, at a level of severity that is experienced regularly by millions of adolescents on school nights, adolescents have worsened mood and decreased ability to regulate negative emotions.
    Journal of Child Psychology and Psychiatry 02/2014; 55(2):180-90. DOI:10.1111/jcpp.12125
  • Journal of Child Psychology and Psychiatry 01/2014; 55(1):96-96. DOI:10.1111/jcpp.12191
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    ABSTRACT: Background Maternal Smoking During Pregnancy (SDP) has consistently been associated with increased risk of attention-deficit/hyperactivity disorder (ADHD) in offspring, but recent studies indicate that this association might be due to unmeasured familial confounding. MethodsA total of 813,030 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Data on maternal SDP and ADHD diagnosis were obtained from national registers and patients were followed up from the age of 3 to the end of 2009. Hazard Ratios (HRs) were estimated using stratified Cox regression models. Cousin and sibling data were used to control for unmeasured familial confounding. ResultsAt the population level maternal SDP predicted ADHD in offspring (HRModerateSDP=1.89; HRHighSDP=2.50). This estimate gradually attenuated toward the null when adjusting for measured confounders (HRModerateSDP=1.62; HRHighSDP=2.04), unmeasured confounders shared within the extended family (i.e., cousin comparison) (HRModerateSDP=1.45; HRHighSDP=1.69), and unmeasured confounders within the nuclear family (i.e., sibling comparison) (HRModerateSDP=0.88; HRHighSDP=0.84). Conclusions Our results suggest that the association between maternal SDP and offspring ADHD are due to unmeasured familial confounding.
    Journal of Child Psychology and Psychiatry 01/2014; 55(1):61-8. DOI:10.1111/jcpp.12124