Journal of Child Psychology and Psychiatry (J CHILD PSYCHOL PSYC)

Publisher: Association for Child Psychology and Psychiatry, Wiley

Journal description

The Journal of Child Psychology and Psychiatry is internationally recognised to be the leading journal covering both child and adolescent psychology and psychiatry. Articles published include experimental and developmental studies, especially those relating to developmental psychopathology and the developmental disorders. An important function of the Journal is to bring together empirical research, clinical studies and reviews of high quality arising from different theoretical perspectives.

Current impact factor: 6.46

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 6.459
2013 Impact Factor 5.669
2012 Impact Factor 5.422
2011 Impact Factor 4.281
2010 Impact Factor 4.36
2009 Impact Factor 4.983
2008 Impact Factor 4.854
2007 Impact Factor 4.432

Impact factor over time

Impact factor

Additional details

5-year impact 6.68
Cited half-life 9.20
Immediacy index 1.21
Eigenfactor 0.03
Article influence 2.50
Website Journal of Child Psychology and Psychiatry and Allied Disciplines, The website
Other titles Journal of child psychology and psychiatry and allied disciplines, Journal of child psychology and psychiatry
ISSN 0021-9630
OCLC 1307942
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 2 years embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • On author's personal website, institutional repositories, arXiv, AgEcon, PhilPapers, PubMed Central, RePEc or Social Science Research Network
    • Author's pre-print may not be updated with Publisher's Version/PDF
    • Author's pre-print must acknowledge acceptance for publication
    • Non-Commercial
    • Publisher's version/PDF cannot be used
    • Publisher source must be acknowledged with citation
    • Must link to publisher version with set statement (see policy)
    • If OnlineOpen is available, BBSRC, EPSRC, MRC, NERC and STFC authors, may self-archive after 12 months
    • If OnlineOpen is available, AHRC and ESRC authors, may self-archive after 24 months
    • Publisher last contacted on 07/08/2014
    • This policy is an exception to the default policies of 'Wiley'
  • Classification

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: This study examined whether relocating from a high-poverty neighborhood to a lower poverty neighborhood as part of a federal housing relocation program (HOPE VI; Housing Opportunities for People Everywhere) had effects on adolescent mental and behavioral health compared to adolescents consistently living in lower poverty neighborhoods. Sociodemographic, risk behavior, and neighborhood data were collected from 592 low-income, primarily African-American adolescents and their primary caregivers. Structured psychiatric interviews were conducted with adolescents. Prerelocation neighborhood, demographic, and risk behavior data were also included. Hierarchical Linear Modeling (HLM) was used to test associations between neighborhood variables and risk outcomes. HLM was used to test whether the effect of neighborhood relocation and neighborhood characteristics might explain differences in sexual risk taking, substance use, and mental health outcomes. Adolescents who relocated of HOPE VI neighborhoods (n = 158) fared worse than control group participants (n = 429) on most self-reported mental health outcomes. The addition of subjective neighborhood measures generally did not substantively change these results. Our findings suggest that moving from a high-poverty neighborhood to a somewhat lower poverty neighborhood is not associated with better mental health and risk behavior outcomes in adolescents. The continued effects of having grown up in a high-poverty neighborhood, the small improvements in their new neighborhoods, the comparatively short length of time they lived in their new neighborhood, and/or the stress of moving appears to worsen most of the mental health outcomes of HOPE VI compared to control group participants who consistently lived in the lower poverty neighborhoods. © 2015 Association for Child and Adolescent Mental Health.
    Journal of Child Psychology and Psychiatry 02/2015; 56(11). DOI:10.1111/jcpp.12386

  • Journal of Child Psychology and Psychiatry 12/2014; 55(12):e6-e6. DOI:10.1111/jcpp.12361
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    ABSTRACT: Background Maternal Smoking During Pregnancy (SDP) has consistently been associated with increased risk of attention-deficit/hyperactivity disorder (ADHD) in offspring, but recent studies indicate that this association might be due to unmeasured familial confounding. MethodsA total of 813,030 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Data on maternal SDP and ADHD diagnosis were obtained from national registers and patients were followed up from the age of 3 to the end of 2009. Hazard Ratios (HRs) were estimated using stratified Cox regression models. Cousin and sibling data were used to control for unmeasured familial confounding. ResultsAt the population level maternal SDP predicted ADHD in offspring (HRModerateSDP=1.89; HRHighSDP=2.50). This estimate gradually attenuated toward the null when adjusting for measured confounders (HRModerateSDP=1.62; HRHighSDP=2.04), unmeasured confounders shared within the extended family (i.e., cousin comparison) (HRModerateSDP=1.45; HRHighSDP=1.69), and unmeasured confounders within the nuclear family (i.e., sibling comparison) (HRModerateSDP=0.88; HRHighSDP=0.84). Conclusions Our results suggest that the association between maternal SDP and offspring ADHD are due to unmeasured familial confounding.
    Journal of Child Psychology and Psychiatry 10/2014; 55(1):61-8. DOI:10.1111/jcpp.12124
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    ABSTRACT: Background: Although long-term treatment is a core aspect of the management of children and adolescents with bipolar disorder (BD), most clinical recommendations are based on results from short-term studies or adult data. In order to guide clinical practice, we review the efficacy and safety profile of mood stabilizers, antipsychotics, and other pharmacological strategies for the long-term treatment of BD in pediatric patients. Methods: A MEDLINE, EMBASE, Cochrane and PsycInfo search (inception through November 2013) was performed to identify prospective studies longer than 12 weeks assessing the use of pharmacological strategies for the long-term treatment of BD in pediatric patients (0-18 years of age). Results: Four randomized controlled trials (RCT) [three placebo-controlled (assessing aripiprazole (2) and flax oil), and one head-to-head comparison of lithium vs. divalproex], and thirteen noncontrolled studies (six open-label studies assessing lithium or anticonvulsants, five assessing second-generation antipsychotics (SGAs) and four assessing combination strategies) were included in the review. Aripiprazole has shown efficacy for relapse prevention in children with pediatric bipolar disorder (PBD) 4-9 years of age in one placebo-controlled RCT. Positive results have been reported in noncontrolled studies with quetiapine and lithium for relapse prevention, as well as with lithium, quetiapine, ziprasidone, and the combination of risperidone and divalproex or lithium for long-term symptom reduction in PBD. The most frequently reported adverse events in children and adolescents treated with lithium and anticonvulsants are gastrointestinal and neurological, whereas use of SGAs is mainly related to weight gain and sedation. Conclusion: According to the limited empirical evidence, aripiprazole can be useful for relapse prevention in children with PBD. Given the lack of consistent efficacy data, clinical decision making should be based on individual clinical aspects and safety concerns.
    Journal of Child Psychology and Psychiatry 06/2014; 55(9). DOI:10.1111/jcpp.12271
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    ABSTRACT: Heterogeneity in the presentation, antecedents, prognosis and treatment response of antisocial behaviour has long provided a challenge to developmental psychopathology researchers. As illustrated in the incisive Frick and colleagues' Annual Research Review, there is growing evidence that the presence of high callous‐unemotional (CU) traits identifies a subgroup of antisocial young people with a particularly aggressive and pervasive form of disorder. Frick and colleagues extend their developmental psychopathology approach to CU traits by linking in theories of conscience development and considering evidence on the stability of CU traits. This commentary addresses these themes and the area more generally, considering (1) comparison of a CU specifier to alternative approaches to antisocial heterogeneity (2) high CU traits in the absence of antisocial behaviour and (3) aspects of the measurement of CU traits.
    Journal of Child Psychology and Psychiatry 06/2014; 55(6). DOI:10.1111/jcpp.12253
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    ABSTRACT: The field of developmental cognitive neuroscience is now established as a discipline at the nexus of the broader fields of developmental psychology and cognitive neuroscience. Sitting in its rear view mirror, but gaining rapidly, is the nascent discipline of developmental social neuroscience. Given the relative youth of this field, it is not surprising that a great deal of energy has gone into generating a rich corpus of empirical data. © 2014 The Authors. Journal of Child Psychology and Psychiatry.
    Journal of Child Psychology and Psychiatry 06/2014; 55(6). DOI:10.1111/jcpp.12254
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    ABSTRACT: We all have a tendency to be easily seduced by Occam's razor, or the desire to accept the simplest hypothesis to explain a given phenomenon. If we observe that children with ADHD come from more impoverished backgrounds or have diets that contain more additives than their peers, then a parsimonious theory would be that poverty and/or diet are causal factors in the development of ADHD. Such theories are all the more attractive because they suggest potential targets for intervention: improve family circumstances and child diet and symptoms of ADHD should diminish. Of course in reality untangling causal relationships is much more complicated. We now accept that many common disorders reflect the confluence of genetic and environmental risk factors, but that any given risk factor is likely to account for only a tiny amount of explained variance in symptom profile. What's more, in order to fulfil the promise of intervention, we must begin to unravel the precise mechanisms by which identified risk factors affect the developing system. This issue showcases two key methodologies for understanding causal influences on developmental disorders: longitudinal designs and well-controlled intervention studies. Both may employ statistical techniques that can identify the mediators of observed associations, elucidating potential mechanistic processes.
    Journal of Child Psychology and Psychiatry 05/2014; 55(5):413-415. DOI:10.1111/jcpp.12239
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    ABSTRACT: Background The relationship between inadequate sleep and mood has been well-established in adults and is supported primarily by correlational data in younger populations. Given that adolescents often experience shortened sleep on school nights, we sought to better understand the effect of experimentally induced chronic sleep restriction on adolescents' mood and mood regulation. Methods Fifty healthy adolescents, ages 14-17, completed a 3-week sleep manipulation protocol involving a baseline week, followed by a sleep restriction (SR) condition (6.5hr in bed per night for five nights) and healthy sleep duration (HS) condition (10hr in bed per night for five nights). The study used a randomized, counterbalanced, crossover experimental design. Participants' sleep was monitored at home via self-report and actigraphy. At the end of each condition, participants and their parents completed questionnaires of mood and mood regulation. To assess for expectancy effects, we also analyzed parent and teen ratings of hyperactivity/impulsivity, which prior research suggests is not sensitive to SR in adolescents. Wilcoxon Signed Rank tests compared questionnaire outcomes across the two conditions. ResultsParticipants averaged 2.5 more hours of sleep per night during HS relative to SR. Compared with HS, adolescents rated themselves as significantly more tense/anxious, angry/hostile, confused, and fatigued, and as less vigorous (p=.001-.01) during SR. Parents and adolescents also reported greater oppositionality/irritability and poorer emotional regulation during SR compared with HS (p<.05). There were no cross-condition differences in depression or hyperactivity/impulsivity (p>.05). Conclusions Findings complement prior correlational study results to show that after only a few days of shortened sleep, at a level of severity that is experienced regularly by millions of adolescents on school nights, adolescents have worsened mood and decreased ability to regulate negative emotions.
    Journal of Child Psychology and Psychiatry 02/2014; 55(2):180-90. DOI:10.1111/jcpp.12125

  • Journal of Child Psychology and Psychiatry 01/2014; 55(1):96-96. DOI:10.1111/jcpp.12191