Journal of Biomechanics (J BIOMECH)

Publisher: University of Michigan. Highway Safety Research Institute; American Society of Biomechanics; European Society of Biomechanics; International Society of Biomechanics; Japanese Society for Clinical Biomechanics and Related Research; All authors, Elsevier

Journal description

The Journal of Biomechanics publishes reports of original and substantial findings using the principles of mechanics to explore biological problems. Analytical, as well as experimental papers may be submitted. Substantially new techniques not testing some explicit hypothesis or reporting original observations may be considered for Technical Notes. The criteria for acceptance of manuscripts include excellence, novelty, significance, clarity, conciseness and interest to the readership. Papers published in the journal may cover a wide range of topics in biomechanics, including, but not limited to: Fundamental Topics - Dynamics of the musculoskeletal system, mechanics of hard and soft tissues, mechanics of muscles, mechanics of bone remodelling, mechanics of implant-tissue interfaces, mechanisms of cells. Cardiovascular and Respiratory Biomechanics - Mechanics of blood flow, air flow, mechanics of the soft tissues, flow-tissue or flow-prosthesis interactions. Dental Biomechanics - Design and analysis of dental prostheses, mechanics of chewing. Injury Biomechanics - Mechanics of impact, dynamics of man-machine interaction. Orthopedic Biomechanics - Mechanics of fracture and fracture fixation, mechanics of implants and implant fixation, mechanics of bones and joints. Rehabilitation Biomechanics - Analyses of gait, mechanics of prosthetics and orthotics. Sports Biomechanics - Mechanical analyses of sports performance. Cell Biomechanics - Relationship of mechanical environment to cells and tissue responses.The journal is affiliated to the American Society of Biomechanics, the International Society of Biomechanics. and the European Society of Biomechanics. The journal is featured in 'Biomechanics World Wide'.

Current impact factor: 2.75

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 2.751
2013 Impact Factor 2.496
2012 Impact Factor 2.716
2011 Impact Factor 2.434
2010 Impact Factor 2.463
2009 Impact Factor 2.657
2008 Impact Factor 2.784
2007 Impact Factor 2.897
2006 Impact Factor 2.542
2005 Impact Factor 2.364
2004 Impact Factor 1.911
2003 Impact Factor 2.005
2002 Impact Factor 1.889
2001 Impact Factor 1.856
2000 Impact Factor 1.474
1999 Impact Factor 1.536
1998 Impact Factor 1.484
1997 Impact Factor 1.461
1996 Impact Factor 1.512
1995 Impact Factor 1.302
1994 Impact Factor 1.548
1993 Impact Factor 1.058
1992 Impact Factor 1.02

Impact factor over time

Impact factor

Additional details

5-year impact 3.16
Cited half-life 9.30
Immediacy index 0.46
Eigenfactor 0.03
Article influence 0.92
Website Journal of Biomechanics website
Other titles Journal of biomechanics
ISSN 0021-9290
OCLC 1754470
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Authors pre-print on any website, including arXiv and RePEC
    • Author's post-print on author's personal website immediately
    • Author's post-print on open access repository after an embargo period of between 12 months and 48 months
    • Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months
    • Author's post-print may be used to update arXiv and RepEC
    • Publisher's version/PDF cannot be used
    • Must link to publisher version with DOI
    • Author's post-print must be released with a Creative Commons Attribution Non-Commercial No Derivatives License
    • Publisher last reviewed on 03/06/2015
  • Classification

Publications in this journal

  • Journal of Biomechanics 11/2015; DOI:10.1016/j.jbiomech.2015.11.041
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    ABSTRACT: The repeated lifting of heavy weights has been identified as a risk factor for low back pain (LBP). Whether squat lifting leads to lower spinal loads than stoop lifting and whether lifting a weight laterally results in smaller forces than lifting the same weight in front of the body remain matters of debate. Instrumented vertebral body replacements (VBRs) were used to measure the in vivo load in the lumbar spine in three patients at level L1 and in one patient at level L3. Stoop lifting and squat lifting were compared in 17 measuring sessions, in which both techniques were performed a total of 104 times. The trunk inclination and amount of knee bending were simultaneously estimated from recorded images. Compared with the aforementioned lifting tasks, the patients additionally lifted a weight laterally with one hand 26 times. Only a small difference (4%) in the measured resultant force was observed between stoop lifting and squat lifting, although the knee-bending angle (stoop 10°, squat 45°) and trunk inclination (stoop 52°, squat 39°) differed considerably at the time points of maximal resultant forces. Lifting a weight laterally caused 14% less implant force on average than lifting the same weight in front of the body. The current in vivo biomechanical study does not provide evidence that spinal loads differ substantially between stoop and squat lifting. The anterior-posterior position of the lifted weight relative to the spine appears to be crucial for spinal loading.
    Journal of Biomechanics 11/2015; DOI:10.1016/j.jbiomech.2015.09.034
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    ABSTRACT: Previous studies have demonstrated distinct clusters of gait patterns in both healthy and pathological groups, suggesting that different movement strategies may be represented. However, these studies have used discrete time point variables and usually focused on only one specific joint and plane of motion. Therefore, the first purpose of this study was to determine if running gait patterns for healthy subjects could be classified into homogeneous subgroups using three-dimensional kinematic data from the ankle, knee, and hip joints. The second purpose was to identify differences in joint kinematics between these groups. The third purpose was to investigate the practical implications of clustering healthy subjects by comparing these kinematics with runners experiencing patellofemoral pain (PFP). A principal component analysis (PCA) was used to reduce the dimensionality of the entire gait waveform data and then a hierarchical cluster analysis (HCA) determined group sets of similar gait patterns and homogeneous clusters. The results show two distinct running gait patterns were found with the main between-group differences occurring in frontal and sagittal plane knee angles (P<0.001), independent of age, height, weight, and running speed. When these two groups were compared to PFP runners, one cluster exhibited greater while the other exhibited reduced peak knee abduction angles (P<0.05). The variability observed in running patterns across this sample could be the result of different gait strategies. These results suggest care must be taken when selecting samples of subjects in order to investigate the pathomechanics of injured runners.
    Journal of Biomechanics 10/2015; 48(14). DOI:10.1016/j.jbiomech.2015.09.025
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    ABSTRACT: In vivo during the day, intervertebral discs are loaded mainly in compression causing fluid and height losses that are subsequently fully recovered overnight due to fluid inflow under smaller compression. However, in vitro, fluid flow through the endplates, in particular fluid imbibition, is hampered possibly by blood clots formed post mortem. Despite earlier in vitro studies, it remains yet unclear if and how fluid flow conditions in vitro could properly emulate those in vivo. Effects of various preload magnitudes (no preload, 0.06 and 0.28MPa) and disc-bone preparation conditions (e.g., w/o bony endplates) on disc height and nucleus pressure were investigated using 54 bovine specimens. Changes in specimen height and pressure at different nucleus locations were used as surrogate measures to assess the fluid content and flow within the discs. Under all investigated preparation conditions and preload magnitudes, no significant pressure recovery could be obtained during low loading phases, even without bony endplates. On the contrary, partial to full displacement recovery were reached in particular under 0.28MPa preload. Results highlight the significant role of disc preload magnitude in disc height recovery during low loading periods. Attention should hence be given in future studies to the proper selection of preload magnitude and duration as well as the animal models used if in vivo response is intended to be replicated. Findings also indicate that flushing the endplates or injection of bone cement respectively neither facilitates nor impedes fluid flow into or out of the disc to a noticeable degree in this bovine disc model.
    Journal of Biomechanics 10/2015; DOI:10.1016/j.jbiomech.2015.10.029
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    ABSTRACT: Lifting is a major risk factor for low back injury. Lifters experience small continual perturbations, because moving a load provides a disturbance to the lifter׳s equilibrium. The goal of the present study was to examine the relationship between local and global trunk/spine stabilities during external perturbations introduced at the foot-floor interface. 12 healthy males were recruited to participate in this study. Participants completed a freestyle lifting protocol on a perturbation treadmill, under three randomized load conditions: ~0, 4, and 8kg. Participants performed a total of 40 lifts under each load condition; no perturbations occurred during the first 20 lifts. During the last 20 lift cycles (in blocks of 5) the participants were randomly perturbed. Local dynamic trunk stability was quantified using the local divergence exponent (λmax) of the first 20 lifts. In addition, the distance traveled from the unperturbed lifting pattern (B), the time to max distance (Tau), the relaxation distance (A), and the rate of return toward the normal lifting pattern (Beta) were analyzed following each external perturbation. An increase in lifted load lead to significantly increased local trunk stability (p=0.046). Higher load also lead to decreased distance (B) traveled away from the unperturbed trajectory (p=0.023). Results agree with previous research that increasing load lifted significantly improves local trunk/spine stability during lifting. Here we have shown that altered local stability also translates into a greater ability to resist external global perturbations, which may reduce injury risk and should be explored in the future.
    Journal of Biomechanics 09/2015; In Press. DOI:10.1016/j.jbiomech.2015.09.026
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    ABSTRACT: Mesenchymal stem cells (MSCs) are the common precursors of several functionally disparate cell lineages. A plethora of chemical and physical stimuli contribute to lineage decisions and guidance, including mechanical stretch concomitant with physical movement. Here, we examined how stretch regulates MSC differentiation into adipocytes and the intracellular signaling pathways involved. MSCs were cultured under adipogenic conditions and divided into a control and an experimental group. Cultures in the experimental group were subjected to a sinusoidal stretch regimen delivered via flexible culture bottoms (5% magnitude, 10 times per min, 6h/day, 3 or 5 days). Expression levels of the adipocyte markers PPARγ-2, adiponectin, and C/EBPα were measured as indices of differentiation. Compared to controls, MSCs exposed to mechanical stretch exhibited downregulated PPARγ-2, adiponectin, and C/EBPα mRNA expression. Alternatively, stretch upregulated phosphorylation of Smad2. This stretch-induced increase in Smad2 phosphorylation was suppressed by pretreatment with the TGFβ1/Smad2 pathway antagonist SB-431542. Pretreatment with the TGFβ1/Smad2 signaling agonist TGFβ1 facilitated the inhibitory effect of stretch on the expression levels of PPARγ-2, adiponectin, and C/EBPα proteins, while pretreatment with SB-431542 reversed the inhibitory effects of subsequent stretch on the expression levels of these markers. These results strongly suggest that the anti-adipogenic effects of mechanical stretch on MSCs are mediated, at least in part, by activation of the TGFβ1/Smad2 signaling pathway. Copyright © 2015. Published by Elsevier Ltd.
    Journal of Biomechanics 08/2015; 48(13). DOI:10.1016/j.jbiomech.2015.08.013
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    ABSTRACT: Biologic tissues respond to the biomechanical conditions to which they are exposed by modifying their architecture. Experimental evidence from the literature suggests that the aim of this process is the mechanical optimization of the tissues (functional adaptation). In particular, this process must produce articular surfaces that, in physiological working conditions, optimize the contact load distribution or, equivalently, maximize the joint congruence. It is thus possible to identify the space of adapted joint configurations (or adapted space of motion) starting solely from knowledge of the shape of the articular surfaces, by determining the envelope of the maximum congruence configurations. The aim of this work was to validate this hypothesis by testing its application on 10 human ankle joints. Digitalizations of articular surfaces were acquired in 10 in-vitro experimental sessions, together with the natural passive tibio-talar motion, which may be considered as representative of the adapted space of motion. This latter was predicted numerically by optimizing the joint congruence. The highest mean absolute errors between each component of predicted and experimental motion were 2.07° and 2.29mm respectively for the three rotations and translations. The present kinematic model replicated the experimentally observed motion well, providing a reliable subject-specific representation of the joint motion starting solely from articulating surface shapes. Copyright © 2015 Elsevier Ltd. All rights reserved.
    Journal of Biomechanics 07/2015; 48(12). DOI:10.1016/j.jbiomech.2015.07.042