Archives of Oral Biology Journal Impact Factor & Information

Publisher: European Organization for Research on Fluorine and Dental Caries Prevention, Elsevier

Journal description

The Archives of Oral Biology publishes papers concerned with advances in knowledge of every aspect of the oral and dental tissues and bone over the whole range of vertebrates, whether from the standpoint of anatomy, bacteriology, biophysics, chemistry, DNA biotechnology, epidemiology, genetics, immunology, molecular biology, palaentology, pathology, physiology or otherwise.

Current impact factor: 1.74

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 1.735
2013 Impact Factor 1.88
2012 Impact Factor 1.549
2011 Impact Factor 1.603
2010 Impact Factor 1.463
2009 Impact Factor 1.649
2008 Impact Factor 1.379
2007 Impact Factor 1.554
2006 Impact Factor 1.655
2005 Impact Factor 1.288
2004 Impact Factor 1.158
2003 Impact Factor 1.098
2002 Impact Factor 1.047
2001 Impact Factor 0.973
2000 Impact Factor 0.845
1999 Impact Factor 1.04
1998 Impact Factor 0.938
1997 Impact Factor 0.735
1996 Impact Factor 0.84
1995 Impact Factor 0.967
1994 Impact Factor 0.959
1993 Impact Factor 0.972
1992 Impact Factor 1.034

Impact factor over time

Impact factor

Additional details

5-year impact 1.89
Cited half-life >10.0
Immediacy index 0.25
Eigenfactor 0.01
Article influence 0.49
Website Archives of Oral Biology website
Other titles Archives of oral biology
ISSN 0003-9969
OCLC 2484813
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Authors pre-print on any website, including arXiv and RePEC
    • Author's post-print on author's personal website immediately
    • Author's post-print on open access repository after an embargo period of between 12 months and 48 months
    • Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months
    • Author's post-print may be used to update arXiv and RepEC
    • Publisher's version/PDF cannot be used
    • Must link to publisher version with DOI
    • Author's post-print must be released with a Creative Commons Attribution Non-Commercial No Derivatives License
    • Publisher last reviewed on 03/06/2015
  • Classification

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: BMP-2 induces osteoblast differentiation and activates osteoclast formation. Here, we investigated the role of Smad1, a molecule that signals downstream of BMP-2, in mediating the effects of BMP-2 on osteoclast differentiation induced by 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) in osteoblasts. The effects of 1,25(OH)2D3 and BMP-2 in osteoclasts were examined using polymerase chain reaction and Western blotting to measure changes in target gene and protein expression. Immunostaining was carried out to investigate the localization of the vitamin D receptor (VDR) in the nucleus in response to BMP-2. Stimulation with both 1,25(OH)2D3 and BMP-2 resulted in significantly greater osteoclast formation and receptor activator of nuclear factor κB ligand (RANKL) mRNA expression compared to stimulation with 1,25(OH)2D3 alone. In addition, expression of the VDR protein was increased, enhancing the activity of 1,25(OH)2D3. Interestingly, knockdown of Smad1 resulted in reduced osteoclast formation, RANKL mRNA expression, and VDR protein expression compared with control cells. Costimulation with 1,25(OH)2D3 and BMP-2 enhanced VDR localization in the nucleus. We found that BMP-2 induced Smad1 activation, thereby influencing the localization of VDR in the nucleus in the presence of 1,25(OH)2D3 and resulting in increased RANKL mRNA expression. These effects ultimately resulted in enhanced osteoclast differentiation. Copyright © 2015 Elsevier Ltd. All rights reserved.
    Archives of Oral Biology 06/2015; 60(9). DOI:10.1016/j.archoralbio.2015.06.001
  • [Show abstract] [Hide abstract]
    ABSTRACT: To evaluate the in vitro effectiveness of APDI with a 660nm laser combined with methylene blue (MB), toluidine blue ortho (TBO) and malachite green (MG) dyes to inactivate Staphylococcus aureus (ATCC 25923) biofilms in compact and cancellous bone specimens. Eighty specimens of compact and 80 of cancellous bone were contaminated with a standard suspension of the microorganism and incubated for 14 days at 37°C to form biofilms. After this period, the specimens were divided into groups (n=10) according to established treatment: PS-L- (control - no treatment); PSmb+L-, PStbo+L-, PSmg+L- (only MB, TBO or MG for 5min in the dark); PS-L+ (only laser irradiation for 180s); and APDImb, APDItbo and APDImg (APDI with MB, TBO or MG for 180s). The findings were statistically analyzed by ANOVA at 5% significance levels. All experimental treatments showed significant reduction of logCFU/mL S. aureus biofilms when compared with the control group for compact and cancellous bones specimens; the APDI group's treatment was more effective. The APDI carried out for the compact specimens showed better results when compared with cancellous specimens at all times of application. For the group of compact bone, APDImg showed greater reductions in CFU/mL (4.46log10). In the group of cancellous bone, the greatest reductions were found in the APDImb group (3.06log10). APDI with methylene blue, toluidine blue ortho and malachite green dyes and a 660nm laser proved to be effective in the inactivation of S. aureus biofilms formed in compact and cancellous bone. Copyright © 2015. Published by Elsevier Ltd.
    Archives of Oral Biology 05/2015; 60(5). DOI:10.1016/j.archoralbio.2015.02.010
  • [Show abstract] [Hide abstract]
    ABSTRACT: The aim of this paper was to compare the chemical composition of human teeth with other mammal species that are likely candidates for replacing them in studies that test dental material. Dentine and enamel fragments extracted from 400 sound human, bovine, porcine and ovine - 100 teeth per species - incisors and molars were mechanically ground up to a final particle size of less than 100μm. C/N analysis, thermogravimetric analysis coupled to mass spectrometry (TG-MS), and wavelength dispersive X-ray fluorescence (WDXRF) were used to analyse the samples' composition. Elemental analysis showed more organic carbon and nitrogen in dentine than in enamel. Human enamel was the most highly mineralised, with C and N values close to hydroxyapatite. Bovine dentine and enamel were the most similar to human. TG-MS: in all species, enamel contained less carbon and organic matter than dentine. Thermal decomposition of human enamel showed great similarity to synthetic hydroxyapatite, and large differences from bovine, ovine and porcine enamel. Thermal decomposition showed the greatest similarity between human and bovine dentine. Dentine contained larger quantities of Mg, S, Sr and Zn than enamel. Enamel contained larger quantities of P, Ca, Cl, Cu, K and Ca/P ratio than dentine. Human enamel and dentine contained a higher Ca/P ratio, larger quantities of Cl and Cu and lower quantities of Mg, S, Zn than the animal species. Elemental analysis, TG-MS and WDXRF have shown that human and bovine enamel and dentine show the greatest similarity among the species analysed. Copyright © 2015 Elsevier Ltd. All rights reserved.
    Archives of Oral Biology 05/2015; 60(5). DOI:10.1016/j.archoralbio.2015.01.014
  • [Show abstract] [Hide abstract]
    ABSTRACT: The aim of this article was to study the variation in oral microflora of the subgingival plaque during and after radiotherapy. During and after radiotherapy, microbial samples were collected at seven time points (early stage, medium stage, and later stage of radiotherapy, and 1 month, 3 months, 6 months, and 1 year after radiotherapy) in three subjects for a total of 21 samples. Polymerase chain reaction (PCR) amplification was carried out on the 16S rDNA hypervariable V1-V3 region, and then the PCR products were determined by high-throughput pyrosequencing. The rarefaction curve indicating the richness of the microflora demonstrated that the number of operational taxonomic units (OTUs) was in decline from the early stage of radiotherapy to the time point 1 month after radiotherapy and then trended upward. The Shannon diversity index declined during radiotherapy (ranging from 4.59 to 3.73), and generally rose after radiotherapy, with the lowest value of 3.5 (1 month after radiotherapy) and highest value of 4.75 (6 months after radiotherapy). A total of 120 genera were found; five genera (Actinomyces, Veillonella, Prevotella, Streptococcus, Campylobacter) were found in all subjects across all time points. The richness and diversity of oral ecology decreased with increased radiation dose, and it was gradually restored with time. Copyright © 2015 Elsevier Ltd. All rights reserved.
    Archives of Oral Biology 05/2015; 60(9). DOI:10.1016/j.archoralbio.2015.05.006
  • [Show abstract] [Hide abstract]
    ABSTRACT: Periodontal disease is one of the most prevalent oral diseases, which is associated with inflammation of the tooth-supporting tissues. Tormentic acid (TA), a triterpene isolated from Rosa rugosa, has been reported to exert anti-inflammatory effects. The aim of this study was to investigate the anti-inflammatory effects of TA on lipopolysaccharide (LPS)-stimulated human gingival fibroblasts (HGFs). The levels of inflammatory cytokines such as interleukin (IL)-6 and chemokines such as IL-8 were detected by enzyme-linked immunosorbent assay (ELISA). The expression of Toll-like receptor 4 (TLR4), nuclear factor kappa B (NF-κB), IκBα, p38, extracellular signal-regulated kinase (ERK), and c-Jun N-terminal kinase (JNK) was determined by Western blotting. The results showed that Porphyromonas gingivalis LPS significantly upregulated the expression of IL-6 and IL-8. TA inhibited the LPS-induced production of IL-6 and IL-8 in a dose-dependent manner. Furthermore, TA inhibited LPS-induced TLR4 expression; NF-κB activation; IκBα degradation; and phosphorylation of ERK, JNK, and P38. TA inhibits the LPS-induced inflammatory response in HGFs by suppressing the TLR4-mediated NF-κB and mitogen-activated protein kinase (MAPK) signalling pathway. Copyright © 2015 Elsevier Ltd. All rights reserved.
    Archives of Oral Biology 05/2015; 60(9). DOI:10.1016/j.archoralbio.2015.05.005